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About This Item
実験式（ヒル表記法）:
C8H20N4O2
CAS番号:
分子量:
204.27
MDL番号:
UNSPSCコード:
12352106
PubChem Substance ID:
NACRES:
NA.25
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輸送温度
dry ice
保管温度
−70°C
SMILES記法
[H][N+]([H])(C)CCCCCCN(C)N([O-])N=O
InChI
1S/C8H19N4O2/c1-9-7-5-3-4-6-8-11(2)12(14)10-13/h9H,3-8H2,1-2H3/q-1/p+1
InChI Key
WASRCWNCCFPNEJ-UHFFFAOYSA-O
アプリケーション
NOドナ-
生物化学的／生理学的作用
NO放出:t1/2 = 3分(リン酸緩衝液、22℃)
保管分類コード
11 - Combustible Solids
WGK
WGK 3
引火点(°F)
Not applicable
引火点(℃)
Not applicable
個人用保護具 (PPE)
Eyeshields, Gloves, type N95 (US)
適用法令
試験研究用途を考慮した関連法令を主に挙げております。化学物質以外については、一部の情報のみ提供しています。 製品を安全かつ合法的に使用することは、使用者の義務です。最新情報により修正される場合があります。WEBの反映には時間を要することがあるため、適宜SDSをご参照ください。
Jan Code
M1555-50MG:
M1555-VAR:
M1555-BULK:
Philip E James et al.
Circulation research, 94(7), 976-983 (2004-02-14)
Vascular dysfunction in diabetes is attributed to lack of bioavailable nitric oxide (NO) and is postulated as a primary cause of small vessel complications as a result of poor glycemic control. Although it has been proposed that NO is bound
D Clark et al.
Molecular plant-microbe interactions : MPMI, 13(12), 1380-1384 (2000-12-06)
We used a variety of nitric oxide (NO) donors to demonstrate that NO inhibits the activities of tobacco catalase and ascorbate peroxidase (APX). This inhibition appears to be reversible because removal of the NO donor led to a significant recovery
Stefan Muenster et al.
Anesthesiology, 125(5), 952-963 (2016-10-19)
Transfusion of packed erythrocytes stored for a long duration is associated with increased pulmonary arterial pressure and vascular resistance. Prolonged storage decreases erythrocyte deformability, and older erythrocytes are rapidly removed from the circulation after transfusion. The authors studied whether treating
H P Monteiro et al.
Free radical biology & medicine, 28(2), 174-182 (2001-04-03)
Nitric oxide (NO) can participate in cellular signaling. In this study, monoclonal antibodies against proteins from the growth factor-mediated signalling pathway were used to identify a set of 126-, 56-, 43-, and 40-kDa proteins phosphorylated on tyrosine at NO stimulation
S H Lin et al.
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism, 20(9), 1380-1391 (2000-09-20)
Neuronal injury may be dependent upon the generation of the free radical nitric oxide (NO) and the subsequent induction of programmed cell death (PCD). Although the nature of this injury may be both preventable and reversible, the underlying mechanisms that
資料
Nitric oxide (NO) as a signal transporter in neurons, endothelial cells and in the immune system.
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