P1742
Prealbumin from human plasma
lyophilized powder
Sinônimo(s):
Thyroxine binding prealbumin, Transthyretin
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About This Item
Produtos recomendados
fonte biológica
human plasma
Nível de qualidade
Ensaio
≥95% (SDS-PAGE)
Formulário
lyophilized powder
técnica(s)
immunoelectrophoresis: suitable
immunoprecipitation (IP): suitable
Impurezas
Infectious agents, tested
solubilidade
H2O: soluble 1 mg/mL
ε (coeficiente de extinção)
13.5 at 280 nm at 1%
nº de adesão UniProt
temperatura de armazenamento
−20°C
Informações sobre genes
human ... TTR(7276)
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Descrição geral
Human prealbumin is a product of chromosome 18. It is a serum protein, synthesized primarily in the liver. It is a tetrameric protein with a molecular weight of 55 kDa. Human prealbumin is composed of four identical non-covalently bound monomers of 127 amino acid residues arranged with tetrahedral symmetry.
Aplicação
Human prealbumin was used to study reduced transthyretin expression in sera of lung cancer.
Prealbumin from human plasma has been used as a positive control in immunoprecipitation as a reference standard in quantitative rocket immunoelectrophoresis for quantification of cerebrospinal fluid.
Ações bioquímicas/fisiológicas
Human prealbumin has been observed in carcinoid tumors.
Prealbumin levels are indictors of malnutrition and may be modulated during inflammation. It is regarded as potential marker of protein energy malnutrition (PEM) during chronic kidney failure supported dialysis. Low levels of prealbumin poses high risk to heart failure (HF).
Embalagem
Package size based on protein content
forma física
Lyophilized powder containing sodium phosphate and NaCl
Código de classe de armazenamento
11 - Combustible Solids
Classe de risco de água (WGK)
WGK 3
Ponto de fulgor (°F)
Not applicable
Ponto de fulgor (°C)
Not applicable
Equipamento de proteção individual
Eyeshields, Gloves, type N95 (US)
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A nationwide study of CMT and FAP has been performed. In FAP TTR Met30 families with late onset, neuropathy showed male preponderance, low penetrance, little relationship to endemic foci, sensorimotor symptoms beginning distally in the lower extremities with disturbance of
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The treatment of familial amyloid polyneuropathies (FAP) is complex and requires a neurological and cardiological multidisciplinary coverage. It includes specific treatments to control the progression of the systemic amyloidogenesis, the symptomatic treatment of the peripheral and autonomic neuropathy (digestive, urinary
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