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Key Documents

AB5898

Sigma-Aldrich

Anti-Protein Gene Product 9.5 Antibody

serum, Chemicon®

Synonim(y):

PGP 9.5, UCH-L1

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About This Item

Kod UNSPSC:
12352203
eCl@ss:
32160702
NACRES:
NA.41

pochodzenie biologiczne

guinea pig

Poziom jakości

forma przeciwciała

serum

rodzaj przeciwciała

primary antibodies

klon

polyclonal

reaktywność gatunkowa

human, rat, mouse

producent / nazwa handlowa

Chemicon®

metody

immunohistochemistry: suitable

numer dostępu NCBI

numer dostępu UniProt

Warunki transportu

dry ice

docelowa modyfikacja potranslacyjna

unmodified

informacje o genach

human ... UCHL1(7345)
mouse ... Uchl1(22223)
rat ... Uchl1(29545)

Specyficzność

Recognizes Protein Gene Product 9.5 (PGP9.5).

Immunogen

A 17 amino acid peptide corresponding to amino acids 175-191 from soluble cytoplasmic human PGP9.5.

Zastosowanie

Anti-Protein Gene Product 9.5 Antibody detects level of Protein Gene Product 9.5 & has been published & validated for use in IH.
Immunohistochemistry: 1:500.

Optimal working dilutions must be determined by end user.

blocking buffer for 1 hour at room temperature. Primary antibody was diluted in blocking buffer to the appropriate working dilution. Blocking buffer was removed and the slides were then incubated at 4°C for 18-24 hours with AB5898 (1:500). After rinsing in PBS 3 times sections were incubated for 60 minutes at room temperature with Cy3-conjugated secondary antibodies. After mounting in a mixture of PBS and glycerol (1:3) containing 0.1% p-phenylenediamine, sections were examined with a Nikon Microphot-SA epifluorescence microscope.
Research Category
Neuroscience
Research Sub Category
Neuronal & Glial Markers

Postać fizyczna

Liquid. Contains 0.05% sodium azide.
Unpurified

Przechowywanie i stabilność

Maintain for 1 year at -20°C from date of shipment. Aliquot to avoid repeated freezing and thawing. For maximum recovery of product, centrifuge the original vial after thawing and prior to removing the cap.

Komentarz do analizy

Control
Brain tissue

Inne uwagi

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

Informacje prawne

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Oświadczenie o zrzeczeniu się odpowiedzialności

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Kod klasy składowania

10 - Combustible liquids

Klasa zagrożenia wodnego (WGK)

WGK 1


Certyfikaty analizy (CoA)

Poszukaj Certyfikaty analizy (CoA), wpisując numer partii/serii produktów. Numery serii i partii można znaleźć na etykiecie produktu po słowach „seria” lub „partia”.

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Dokumenty związane z niedawno zakupionymi produktami zostały zamieszczone w Bibliotece dokumentów.

Odwiedź Bibliotekę dokumentów

Enhanced insulin clearance in mice lacking TRPM8 channels.
McCoy, DD; Zhou, L; Nguyen, AK; Watts, AG; Donovan, CM; McKemy, DD
American Journal of Physiology. Endocrinology and Metabolism null
Nociceptin effect on intestinal motility depends on opioid-receptor like-1 receptors and nitric oxide synthase co-localization.
Sibaev, A; Fichna, J; Saur, D; Yuece, B; Timmermans, JP; Storr, M
World journal of gastrointestinal pharmacology and therapeutics null
The effect of inflammation on the expression and distribution of the MAS-related gene receptors MrgE and MrgF in the murine ileum.
Avula LR, Buckinx R, Alpaerts K, Costagliola A, Adriaensen D, Van Nassauw L, Timmermans JP.
Histochemistry and Cell Biology null
Serotonin-immunoreactive neurons and mast cells in the mouse esophagus suggest involvement of serotonin in both motility control and neuroimmune interactions.
Hempfling C, Neuhuber WL, Worl J
Neurogastroenterology and Motility null
M Bagyánszki et al.
Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society, 23(6), e237-e248 (2011-04-08)
There are indications that alterations in the nitric oxide (NO) system of relaxation mediate gastrointestinal motor disturbances induced by chronic alcohol consumption (CAC). As CAC is known to inhibit the motility of the mouse small intestine, we investigated in this

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