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Key Documents

N6530

Sigma-Aldrich

N1 Medium Supplement (100×)

solution, sterile-filtered, suitable for cell culture

Sinonimo/i:

N1 Supplement

Autenticatiper visualizzare i prezzi riservati alla tua organizzazione & contrattuali


About This Item

Codice UNSPSC:
12352207
NACRES:
NA.77

Sterilità

sterile-filtered

Livello qualitativo

Forma fisica

solution

tecniche

cell culture | mammalian: suitable

Impurezze

endotoxin, tested

Temperatura di conservazione

2-8°C

Descrizione generale

N1 Medium Supplement (100×) can be used as a supplement to produce complete N1 neural cell growth media or closely related media.

Applicazioni

N1 Medium Supplement (100×) has been used as:
  • a component of the proliferation medium for culturing oligodendrocyte-like cells and neuroblastoma cells
  • a supplement in the Dulbecco′s modified Eagle medium (DMEM/F12) to culture human oligodendrocytes
  • a component of the α-minimum essential medium (MEM) for culturing induced primary retinal pigment epithelium (ipRPE) monolayers

Altre note

Contains 0.5mg/ml recombinant human insulin, 0.5mg/ml human transferrin (partially iron-saturated), 0.5μg/ml sodium selenite, 1.6mg/ml putrescine, and 0.73μg/ml progesterone.

Nota sulla preparazione

Prepared in Earle′s Balanced Salt Solution (EBSS) without phenol red.

Prodotti correlati

N° Catalogo
Descrizione
Determinazione del prezzo

Codice della classe di stoccaggio

12 - Non Combustible Liquids

Classe di pericolosità dell'acqua (WGK)

WGK 1

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable

Dispositivi di protezione individuale

Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)


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Ludmila A Voloboueva et al.
Investigative ophthalmology & visual science, 46(11), 4302-4310 (2005-10-27)
To determine whether (R)-alpha-lipoic acid (LA) protects cultured human fetal retinal pigment epithelial (hfRPE) cells against oxidative injury and identify the pathways that may mediate protection. Cultured hfRPE cells were pretreated with various concentrations of LA for 14 to 16
Miguel Flores-Bellver et al.
Journal of extracellular vesicles, 10(13), e12165-e12165 (2021-11-10)
Age-related macular degeneration (AMD) is a leading cause of blindness worldwide. Drusen are key contributors to the etiology of AMD and the ability to modulate drusen biogenesis could lead to therapeutic strategies to slow or halt AMD progression. The mechanisms
Michael Craigie et al.
Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology, 13(2), 126-142 (2017-11-22)
JC virus (JCV) is a human polyomavirus and the etiologic agent of the demyelinating disease progressive multifocal leukoencephalopathy (PML). PML is observed in patients with underlying immunocompromising conditions, suggesting that neuro-immune interactions between peripheral immune cells and neuro-glia play an
Tomasz M Stępkowski et al.
Cellular and molecular neurobiology, 37(6), 1055-1066 (2016-11-11)
Perturbations in the transport of mitochondria and their quality control in neuronal cells underlie many types of neurological pathologies, whereas systems enabling convenient analysis of mitochondria behavior in cellular models of neurodegenerative diseases are limited. In this study, we present
Lucia Lichvarova et al.
Scientific reports, 9(1), 10603-10603 (2019-07-25)
Forebrain glial cells - ependymal cells and astrocytes -acquire upon injury- a "reactive" phenotype associated with parvalbumin (PV) upregulation. Since free radicals, e.g. reactive oxygen species (ROS) play a role in the pathogenesis of multiple sclerosis, and that PV-upregulation in

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