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225517

Sigma-Aldrich

Methoxylamine hydrochloride solution

25-30 wt. % in H2O

Sinonimo/i:

O-Methylhydroxylamine hydrochloride

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About This Item

Formula condensata:
CH3ONH2 · HCl
Numero CAS:
593-56-6
Peso molecolare:
83.52
Beilstein:
3589723
Numero MDL:
MFCD00012951
Codice UNSPSC:
12352100
ID PubChem:
24853502
NACRES:
NA.22
Stato:
liquid

Stato

liquid

Livello qualitativo

100

Concentrazione

25-30 wt. % in H2O

Indice di rifrazione

n20/D 1.4021

Solubilità

alcohol: soluble(lit.)
water: soluble(lit.)

Densità

1.1 g/mL at 25 °C

Stringa SMILE

Cl[H].CON

InChI

1S/CH5NO.ClH/c1-3-2;/h2H2,1H3;1H
XNXVOSBNFZWHBV-UHFFFAOYSA-N

Applicazioni

Methoxylamine hydrochloride was used as an derivatization agent during the structural investigation of novel epoxyisoprostane phospholipid in mildly oxidized low density lipoprotien.

Avvertenze

Danger

Indicazioni di pericolo

H302,H314,H317,H351,H372,H410

Classi di pericolo

Acute Tox. 4 Oral - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 2 - Eye Dam. 1 - Skin Corr. 1 - Skin Sens. 1 - STOT RE 1

Codice della classe di stoccaggio

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

Classe di pericolosità dell'acqua (WGK)

WGK 3

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable

Dispositivi di protezione individuale

Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter

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Certificati d'analisi (COA)

Lot/Batch Number
STBF1292V
2205AM
07716HN
06701JQ
10916DQ

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A D Watson et al.
The Journal of biological chemistry, 274(35), 24787-24798 (1999-08-24)
One of the earliest steps in the development of the atherosclerotic lesion is the accumulation of monocyte/macrophages within the vessel wall. Oxidized lipids present in minimally modified-low density lipoproteins (MM-LDL) contribute to this process by activating endothelial cells to express
Melissa L Fishel et al.
Molecular aspects of medicine, 28(3-4), 375-395 (2007-06-15)
With our growing understanding of the pathways involved in cell proliferation and signaling, targeted therapies, in the treatment of cancer are entering the clinical arena. New and emerging targets are proteins involved in DNA repair pathways. Inhibition of various proteins
Timothy J Kinsella
Clinical cancer research : an official journal of the American Association for Cancer Research, 15(6), 1853-1859 (2009-02-26)
DNA damage processing by mismatch repair (MMR) and/or base excision repair (BER) can determine the therapeutic index following treatment of human cancers using radiation therapy and several classes of chemotherapy drugs. Over the last decade, basic and translational cancer research
Yanlin Jiang et al.
DNA repair, 8(11), 1273-1282 (2009-09-04)
Oxidative DNA damage has been implicated in a number of central nervous system pathologies. The base excision repair (BER) pathway is one of the most important cellular protection mechanisms that respond to oxidative DNA damage. Human apurinic (apyrimidinic) endonuclease/redox effector
Eva M Goellner et al.
Cancer research, 71(6), 2308-2317 (2011-03-17)
Glioblastoma multiforme (GBM) is a devastating brain tumor with poor prognosis and low median survival time. Standard treatment includes radiation and chemotherapy with the DNA alkylating agent temozolomide (TMZ). However, a large percentage of tumors are resistant to the cytotoxic
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