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Key Documents

L7527

Sigma-Aldrich

Lipoprotein, very low density from human plasma

≥95% (SDS-PAGE), solution

Sinônimo(s):

Pre-β-lipoprotein, VLDL, Very low density lipoprotein

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About This Item

Número CAS:
308068-14-6
Número MDL:
MFCD00164409
Código UNSPSC:
12352211
NACRES:
NA.25

fonte biológica

human plasma

Nível de qualidade

200

Ensaio

≥95% (SDS-PAGE)

forma

solution

nº de adesão UniProt

P02649
P02655
P04114

grupo funcional

ester
phospholipid

Condições de expedição

wet ice

temperatura de armazenamento

2-8°C

Informações sobre genes

human ... APOB(338) , APOC2(344) , APOE(348)

Procurando produtos similares? Visita Guia de comparação de produtos

Ações bioquímicas/fisiológicas

VLDL (Very low density lipoprotein) may be used to study the mechanisms and effects of very low density receptors (VLDLR). VLDL is used as a potential source of fatty acids that support cardiac energy metabolism. VLDL is often studied to differentiate its effects from those of chylomicrons.
Inhibits DNA synthesis in lymphocytes activated by the nonspecific mitogen concanavalin A (Con A).

forma física

Solution in 150 mM NaCl and 0.01% EDTA, pH 7.4

Outras notas

View more information on lipoprotein function and lipid transport at www.sigma-aldrich.com/enzymeexplorer

Código de classe de armazenamento

12 - Non Combustible Liquids

Classe de risco de água (WGK)

WGK 2

Ponto de fulgor (°F)

Not applicable

Ponto de fulgor (°C)

Not applicable

Certificados de análise (COA)

Busque Certificados de análise (COA) digitando o Número do Lote do produto. Os números de lote e remessa podem ser encontrados no rótulo de um produto após a palavra “Lot” ou “Batch”.

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Resistin-like molecule (RELM) β is a secretory protein homologous to resistin and reportedly contributes to local immune response regulation in gut and bronchial epithelial cells. However, we found that activated macrophages also express RELMβ and thus investigated the role of
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Since Zilversmit first proposed postprandial lipemia as the most common risk of cardiovascular disease, chylomicrons (CM) and CM remnants have been thought to be the major lipoproteins which are increased in the postprandial hyperlipidemia. However, it has been shown over
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Analytical and bioanalytical chemistry, 406(25), 6203-6211 (2014-08-12)
High-performance affinity chromatography (HPAC) was utilized to examine the binding of very low density lipoprotein (VLDL) with drugs, using R/S-propranolol as a model. These studies indicated that two mechanisms existed for the binding of R- and S-propranolol with VLDL. The
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Journal of lipids, 2011, 189876-189876 (2011-07-21)
The heart is a major consumer of energy and is able to utilise a wide range of substrates including lipids. Nonesterified fatty acids (NEFA) were thought to be a favoured carbon source, but their quantitative contribution is limited because of
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Biochemical and biophysical research communications, 285(2), 393-399 (2001-07-11)
Human very-low-density lipoprotein (VLDL) inhibits DNA synthesis in lymphocytes activated by the nonspecific mitogen concanavalin A (Con A). We studied the effects of VLDL on lymphocyte activation (IL-2 receptor expression), cell cycle progression, and production of IL-2 and of IL-4
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