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SAB5300197

Sigma-Aldrich

Monoclonal Anti-TGFB1 antibody produced in mouse

clone 4F9C10, ascites fluid

Synonym(s):

CED, DPD1, TGFB, TGFB1, TGFbeta

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

biological source

mouse

conjugate

unconjugated

antibody form

ascites fluid

antibody product type

primary antibodies

clone

4F9C10, monoclonal

species reactivity

human

technique(s)

direct ELISA: 1:10,000
western blot: 1:500-1:2,000

isotype

IgG2a

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... TGF beta1(7040)

Immunogen

Purified recombinant fragment of TGF beta1 expressed in E.coli.
Mouse monoclonal antibody raised against TGF beta1

Physical form

Ascitic fluid containing 0.03% sodium azide.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class

10 - Combustible liquids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Yu-Cheng Chin et al.
Journal of nanobiotechnology, 20(1), 373-373 (2022-08-12)
The escape of bladder cancer from immunosurveillance causes monotherapy to exhibit poor efficacy; therefore, designing a multifunctional nanoparticle that boosts programmed cell death and immunoactivation has potential as a treatment strategy. Herein, we developed a facile one-pot coprecipitation reaction to
Akrivi Chrysanthopoulou et al.
The Journal of pathology, 233(3), 294-307 (2014-04-18)
Neutrophil activation by inflammatory stimuli and the release of extracellular chromatin structures (neutrophil extracellular traps - NETs) have been implicated in inflammatory disorders. Herein, we demonstrate that NETs released by neutrophils treated either with fibrosis-related agents, such as cigarette smoke
Liqing Xu et al.
Cellular immunology, 290(1), 89-95 (2014-06-21)
Although CD4(+)CD25(+) Treg (Treg) cells are known to modulate NK cell functions, the modulation mechanism of these cells in cord blood has not been fully clarified. The purpose of this study was to clarify the mechanism whereby cord blood Treg

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