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EM0200

Sigma-Aldrich

Osteo-Bed Bone Embedding Kit

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About This Item

UNSPSC Code:
12171500
NACRES:
NA.47

Application

Suitable for use with large and small mineralized (undecalcified) bone sections. Yields clear, hard blocks for cutting sections. Not water-soluble.

Recommended products

Osteo-Bed Bone Embedding Solvent (O8639) is available to remove plastic from sections.

Kit Components Only

Product No.
Description

  • Osteo-Bed Bone Embedding Resin A (O8514) 900 mL

  • Osteo-Bed Bone Embedding Catalyst (O8764) 2 x 12

recommended

Product No.
Description
Pricing

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Danger

Hazard Classifications

Aquatic Acute 1 - Aquatic Chronic 1 - Eye Irrit. 2 - Flam. Liq. 2 - Org. Perox. D - Repr. 1B - Skin Irrit. 2 - Skin Sens. 1 - STOT SE 3

target_organs

Respiratory system

Storage Class

5.2 - Organic peroxides and self-reacting hazardous materials

flash_point_f

50.0 °F

flash_point_c

10 °C


Certificates of Analysis (COA)

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Colin K Yee et al.
Clinical & experimental metastasis, 25(8), 903-911 (2008-09-25)
In the current study, we examine heparin's anti-metastatic properties by using a well-defined mouse model of osteolytic bone metastasis. C57BL/6 mice were treated with increasing doses of unfractionated heparin (15, 20, or 25 units/mouse) 30 min prior to the left
Nur-Vaizura Mohamad et al.
International journal of medical sciences, 18(16), 3665-3673 (2021-11-19)
Menopause is the leading cause of osteoporosis for elderly women due to imbalanced bone remodelling in the absence of oestrogen. The ability of tocotrienol in reversing established bone loss due to oestrogen deficiency remains unclear despite the plenitude of evidence
Xuekun Fu et al.
Signal transduction and targeted therapy, 5(1), 297-297 (2020-12-29)
In vertebrates, the type 1 parathyroid hormone receptor (PTH1R) is a critical regulator of skeletal development and homeostasis; however, how it is modulated is incompletely understood. Here we report that deleting Kindlin-2 in osteoblastic cells using the mouse 10-kb Dmp1-Cre
Yishu Wang et al.
JCI insight, 4(22) (2019-11-15)
Mammalian focal adhesion proteins Pinch1 and Pinch2 regulate integrin activation and cell-extracellular matrix adhesion and migration. Here, we show that deleting Pinch1 in osteocytes and mature osteoblasts using the 10-kb mouse Dmp1-Cre and Pinch2 globally (double KO; dKO) results in

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