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Key Documents

L7527

Sigma-Aldrich

Lipoprotein, very low density from human plasma

≥95% (SDS-PAGE), solution

Sinonimo/i:

Pre-β-lipoprotein, VLDL, Very low density lipoprotein

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About This Item

Numero CAS:
308068-14-6
Numero MDL:
MFCD00164409
Codice UNSPSC:
12352211
NACRES:
NA.25

Origine biologica

human plasma

Livello qualitativo

200

Saggio

≥95% (SDS-PAGE)

Forma fisica

solution

N° accesso UniProt

P02649
P02655
P04114

Gruppo funzionale

ester
phospholipid

Condizioni di spedizione

wet ice

Temperatura di conservazione

2-8°C

Informazioni sul gene

human ... APOB(338) , APOC2(344) , APOE(348)

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Azioni biochim/fisiol

VLDL (Very low density lipoprotein) may be used to study the mechanisms and effects of very low density receptors (VLDLR). VLDL is used as a potential source of fatty acids that support cardiac energy metabolism. VLDL is often studied to differentiate its effects from those of chylomicrons.
Inhibits DNA synthesis in lymphocytes activated by the nonspecific mitogen concanavalin A (Con A).

Stato fisico

Solution in 150 mM NaCl and 0.01% EDTA, pH 7.4

Altre note

View more information on lipoprotein function and lipid transport at www.sigma-aldrich.com/enzymeexplorer

Codice della classe di stoccaggio

12 - Non Combustible Liquids

Classe di pericolosità dell'acqua (WGK)

WGK 2

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable

Certificati d'analisi (COA)

Cerca il Certificati d'analisi (COA) digitando il numero di lotto/batch corrispondente. I numeri di lotto o di batch sono stampati sull'etichetta dei prodotti dopo la parola ‘Lotto’ o ‘Batch’.

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Sigma-Aldrich

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Resistin-like molecule (RELM) β is a secretory protein homologous to resistin and reportedly contributes to local immune response regulation in gut and bronchial epithelial cells. However, we found that activated macrophages also express RELMβ and thus investigated the role of
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Since Zilversmit first proposed postprandial lipemia as the most common risk of cardiovascular disease, chylomicrons (CM) and CM remnants have been thought to be the major lipoproteins which are increased in the postprandial hyperlipidemia. However, it has been shown over
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Analytical and bioanalytical chemistry, 406(25), 6203-6211 (2014-08-12)
High-performance affinity chromatography (HPAC) was utilized to examine the binding of very low density lipoprotein (VLDL) with drugs, using R/S-propranolol as a model. These studies indicated that two mechanisms existed for the binding of R- and S-propranolol with VLDL. The
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The heart is a major consumer of energy and is able to utilise a wide range of substrates including lipids. Nonesterified fatty acids (NEFA) were thought to be a favoured carbon source, but their quantitative contribution is limited because of
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Biochemical and biophysical research communications, 285(2), 393-399 (2001-07-11)
Human very-low-density lipoprotein (VLDL) inhibits DNA synthesis in lymphocytes activated by the nonspecific mitogen concanavalin A (Con A). We studied the effects of VLDL on lymphocyte activation (IL-2 receptor expression), cell cycle progression, and production of IL-2 and of IL-4
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