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Documentos Principais

SAB5300267

Sigma-Aldrich

Monoclonal Anti-Cytokeratin 5 antibody produced in mouse

clone 2C2, purified immunoglobulin, buffered aqueous solution

Sinônimo(s):

DDD, EBS2, K5, KRT5, KRT5A

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About This Item

Código UNSPSC:
12352203
NACRES:
NA.41

fonte biológica

mouse

Nível de qualidade

100

conjugado

unconjugated

forma do anticorpo

purified immunoglobulin

tipo de produto de anticorpo

primary antibodies

clone

2C2, monoclonal

forma

buffered aqueous solution

peso molecular

62.3 kDa

reatividade de espécies

human, mouse, monkey

técnica(s)

direct ELISA: 1:10,000
flow cytometry: 1:200-1:400
immunohistochemistry: 1:200-1:1,000
indirect immunofluorescence: 1:200-1:1,000
western blot: 1:500-1:2,000

Isotipo

IgG1

nº de adesão NCBI

NM_000424

Condições de expedição

wet ice

temperatura de armazenamento

−20°C

modificação pós-traducional do alvo

unmodified

Informações sobre genes

human ... CK5(3852)

Imunogênio

Purified recombinant fragment of human CK5 expressed in E.coli.
Mouse monoclonal antibody raised against CK5

forma física

Purified antibody in PBS with 0.05% sodium azide.

Exoneração de responsabilidade

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Código de classe de armazenamento

10 - Combustible liquids

Classe de risco de água (WGK)

WGK 3

Ponto de fulgor (°F)

Not applicable

Ponto de fulgor (°C)

Not applicable

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Molecular carcinogenesis, 53(10), 841-846 (2013-04-30)
Lung adenocarcinoma (AdC) and lung squamous cell carcinoma (SCC) are the most common non-small cell lung cancer (NSCLC) subtypes, however, most genetic mouse models of lung cancer produce predominantly, if not exclusively, AdC. Whether this is secondary to targeting mutations
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We have witnessed significant progress in gene-based approaches to cancer prognostication, promising early intervention for high-risk patients and avoidance of overtreatment for low-risk patients. However, there has been less advancement in protein-based approaches, even though perturbed protein levels and post-translational
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