90C2
Leukocyte Naphthol AS-D Chloroacetate (Specific Esterase) Kit
Sinônimo(s):
Leder Stain
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About This Item
Código UNSPSC:
12352106
NACRES:
NA.47
Produtos recomendados
descrição
Select reagents packaged in gelatin capsules
Nível de qualidade
prazo de validade
Expiry date on the label.
IVD
for in vitro diagnostic use
aplicação(ões)
hematology
histology
Condições de expedição
wet ice
temperatura de armazenamento
2-8°C
Aplicação
Esterases are ubiquitous in nature and encompass a series of different enzymes acting upon select substrates. With the 90C2 kit, Naphthol AS-D Chloroacetate is enzymatically hydrolyzed by "specific esterase, " liberating a free naphthol compound. This then couples with a diazonium salt, forming highly colored deposits at sites of enzyme activity. This enzyme is usually considered specific for cells of granulocytic lineage. May be used to detect neutrophils in peripheral blood, bone marrow or tissue sections which have been paraffin embedded.
Somente componentes do kit
Nº do produto
Descrição
- Dimethyl formamide (9010) 25 mL
- Fast Corinth V Salt (9015) 10 capsules
Palavra indicadora
Danger
Frases de perigo
Declarações de precaução
Classificações de perigo
Acute Tox. 4 Dermal - Acute Tox. 4 Inhalation - Acute Tox. 4 Oral - Carc. 1B - Eye Dam. 1 - Met. Corr. 1 - Repr. 1B - Skin Corr. 1B
Perigos de suplementos
Código de classe de armazenamento
6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects
Ponto de fulgor (°F)
Not applicable
Ponto de fulgor (°C)
Not applicable
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Journal of immunology (Baltimore, Md. : 1950), 179(11), 7820-7826 (2007-11-21)
Right ventricular (RV) dysfunction is a strong risk factor for poor clinical outcome following pulmonary embolism (PE), the third most prevalent cardiovascular disease. Previous studies in our laboratory demonstrated that RV failure during PE is mediated, in part, by neutrophil-dependant
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Molecular medicine reports, 22(1), 483-493 (2020-04-23)
Inactivation of the Hippo pathway protects the myocardium from cardiac ischemic injury. MicroRNAs (miRs) have been reported to play pivotal roles in the progression of myocardial infarction (MI). The present study examined whether miR‑93 could promote angiogenesis and attenuate remodeling
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