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Key Documents

L7527

Sigma-Aldrich

Lipoprotein, very low density from human plasma

≥95% (SDS-PAGE), solution

Synonim(y):

Pre-β-lipoprotein, VLDL, Very low density lipoprotein

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About This Item

Numer CAS:
308068-14-6
Numer MDL:
MFCD00164409
Kod UNSPSC:
12352211
NACRES:
NA.25

pochodzenie biologiczne

human plasma

Poziom jakości

200

Próba

≥95% (SDS-PAGE)

Postać

solution

numer dostępu UniProt

P02649
P02655
P04114

grupa funkcyjna

ester
phospholipid

Warunki transportu

wet ice

temp. przechowywania

2-8°C

informacje o genach

human ... APOB(338) , APOC2(344) , APOE(348)

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Działania biochem./fizjol.

VLDL (Very low density lipoprotein) may be used to study the mechanisms and effects of very low density receptors (VLDLR). VLDL is used as a potential source of fatty acids that support cardiac energy metabolism. VLDL is often studied to differentiate its effects from those of chylomicrons.
Inhibits DNA synthesis in lymphocytes activated by the nonspecific mitogen concanavalin A (Con A).

Postać fizyczna

Solution in 150 mM NaCl and 0.01% EDTA, pH 7.4

Inne uwagi

View more information on lipoprotein function and lipid transport at www.sigma-aldrich.com/enzymeexplorer
This page may contain text that has been machine translated.

Kod klasy składowania

12 - Non Combustible Liquids

Klasa zagrożenia wodnego (WGK)

WGK 2

Temperatura zapłonu (°F)

Not applicable

Temperatura zapłonu (°C)

Not applicable

Certyfikaty analizy (CoA)

Poszukaj Certyfikaty analizy (CoA), wpisując numer partii/serii produktów. Numery serii i partii można znaleźć na etykiecie produktu po słowach „seria” lub „partia”.

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Arteriosclerosis, thrombosis, and vascular biology, 33(8), 1986-1993 (2013-05-25)
Resistin-like molecule (RELM) β is a secretory protein homologous to resistin and reportedly contributes to local immune response regulation in gut and bronchial epithelial cells. However, we found that activated macrophages also express RELMβ and thus investigated the role of
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Since Zilversmit first proposed postprandial lipemia as the most common risk of cardiovascular disease, chylomicrons (CM) and CM remnants have been thought to be the major lipoproteins which are increased in the postprandial hyperlipidemia. However, it has been shown over
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High-performance affinity chromatography (HPAC) was utilized to examine the binding of very low density lipoprotein (VLDL) with drugs, using R/S-propranolol as a model. These studies indicated that two mechanisms existed for the binding of R- and S-propranolol with VLDL. The
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The heart is a major consumer of energy and is able to utilise a wide range of substrates including lipids. Nonesterified fatty acids (NEFA) were thought to be a favoured carbon source, but their quantitative contribution is limited because of
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Human very-low-density lipoprotein (VLDL) inhibits DNA synthesis in lymphocytes activated by the nonspecific mitogen concanavalin A (Con A). We studied the effects of VLDL on lymphocyte activation (IL-2 receptor expression), cell cycle progression, and production of IL-2 and of IL-4
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