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32-0290

Urea

Name: Urea
Brand: SAJ

SAJ first grade, ≥98.0%

Synonim(y):

Carbamide, Carbonyldiamide

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About This Item

Wzór liniowy:

NH₂CONH₂

Numer CAS:

57-13-6

Masa cząsteczkowa:

60.06

Beilstein:

635724

Numer MDL:

MFCD00008022

Kod UNSPSC:

12352111

Identyfikator substancji w PubChem:

329753970

Polecane produkty

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Sigma-Aldrich

M6250

2-Mercaptoethanol

klasa czystości

SAJ first grade

Próba

≥98.0%

Postać

solid

dostępność

available only in Japan

mp

132-135 °C (lit.)

gęstość

1.335 g/mL at 25 °C (lit.)

ciąg SMILES

NC(N)=O

InChI

1S/CH4N2O/c2-1(3)4/h(H4,2,3,4)

Klucz InChI

XSQUKJJJFZCRTK-UHFFFAOYSA-N

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Zastosowanie

Used for the denaturation of proteins and as a mild solubilization agent for insoluble or denatured proteins. Useful for renaturing proteins from samples already denatured with 6 M guanidine chloride such as inclusion bodies. May be used with guanidine hydrochloride and dithiothreitrol (DTT) in the refolding of denatured proteins into their native or active form.

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Kod klasy składowania

11 - Combustible Solids

Klasa zagrożenia wodnego (WGK)

WGK 1

Temperatura zapłonu (°F)

Not applicable

Temperatura zapłonu (°C)

Not applicable

Certyfikaty analizy (CoA)

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Designing chitosan based eco-friendly multifunctional soil conditioner systems with urea controlled release and water retention.

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Carbohydrate polymers, 223, 115040-115040 (2019-08-21)

The paper reports new soil conditioner systems obtained by in situ hydrogelation of chitosan with salicylaldehyde in the presence of urea fertilizer, designed to address both fertilization and water retention of the soil. The new systems were structural, supramolecular and

Aberrant expression and distribution of enzymes of the urea cycle and other ammonia metabolizing pathways in dogs with congenital portosystemic shunts.

Giora van Straten et al.

PloS one, 9(6), e100077-e100077 (2014-06-20)

The detoxification of ammonia occurs mainly through conversion of ammonia to urea in the liver via the urea cycle and glutamine synthesis. Congenital portosystemic shunts (CPSS) in dogs cause hyperammonemia eventually leading to hepatic encephalopathy. In this study, the gene

Expression of progerin in aging mouse brains reveals structural nuclear abnormalities without detectible significant alterations in gene expression, hippocampal stem cells or behavior.

Jean-Ha Baek et al.

Human molecular genetics, 24(5), 1305-1321 (2014-10-26)

Hutchinson-Gilford progeria syndrome (HGPS) is a segmental progeroid syndrome with multiple features suggestive of premature accelerated aging. Accumulation of progerin is thought to underlie the pathophysiology of HGPS. However, despite ubiquitous expression of lamin A in all differentiated cells, the

Incomplete Restoration of Angiotensin II-Induced Renal Extracellular Matrix Deposition and Inflammation Despite Complete Functional Recovery in Rats.

Anne-Roos S Frenay et al.

PloS one, 10(6), e0129732-e0129732 (2015-06-11)

Some diseases associated with a temporary deterioration in kidney function and/or development of proteinuria show an apparently complete functional remission once the initiating trigger is removed. While it was earlier thought that a transient impairment of kidney function is harmless

Golgi fragmentation and sphingomyelin transport to Chlamydia trachomatis during penicillin-induced persistence do not depend on the cytosolic presence of the chlamydial protease CPAF.

Stephanie Dille et al.

PloS one, 9(7), e103220-e103220 (2014-07-30)

Chlamydia grows inside a cytosolic vacuole (the inclusion) that is supplied with nutrients by the host through vesicular and non-vesicular transport. It is unclear in many respects how Chlamydia organizes this transport. One model posits that the Chlamydia-induced fragmentation of

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