推荐产品
product name
聚-DL-赖氨酸 氢溴酸盐, mol wt >40,000
形狀
powder
分子量
>40,000
技術
cell culture | mammalian: suitable
顏色
white to off-white
儲存溫度
−20°C
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相关类别
分析報告
Molecular weight based on viscosity. Also assayed by MALLS.
其他說明
有关聚氨基酸的其他技术信息，请访问 聚氨基酸常见问题解答资源。
儲存類別代碼
11 - Combustible Solids
水污染物質分類（WGK）
WGK 3
閃點（°F）
Not applicable
閃點（°C）
Not applicable
個人防護裝備
Eyeshields, Gloves, type N95 (US)
其他客户在看
I Bleiberg et al.
Biochimica et biophysica acta, 674(3), 345-353 (1981-05-18)
Heparin uptake by cultured macrophages was investigated from the standpoint of: (1) whether the increased uptake in the presence of polycations is due to charge neutralization, and (2) whether the heparin becomes internalized. Regarding the first point, our results are
L J Anghileri et al.
Tumori, 76(3), 217-219 (1990-06-30)
Treatment of P388 leukemia cells with poly-DL-lysine (Poly-lys) considerably increases the binding of colloidal chromic phosphate (32P). This augmentation of the number of particles that are bound is in direct relationship with Poly-lys concentration, and very significantly with its degree
Xiaogang Liu et al.
Science (New York, N.Y.), 307(5716), 1763-1766 (2005-03-19)
We present a method for controlling the initiation and kinetics of polymer crystal growth using dip-pen nanolithography and an atomic force microscope tip coated with poly-dl-lysine hydrobromide. Triangular prisms of the polymer epitaxially grow on freshly cleaved mica substrates, and
L J Anghileri et al.
Biochimica et biophysica acta, 982(2), 307-308 (1989-07-10)
Colloidal [51Cr]chromic phosphate uptake is considerably increased by preincubation of P388 ascites leukemia cells with poly(DL-lysine). The uptake increase is in direct relationship with the concentration and the degree of polymerization of poly(DL-lysine). The probable implication of cell surface electrical
Jia-Rong Liu et al.
Biochemical pharmacology, 163, 458-471 (2019-03-20)
Glioblastoma is the most fatal type of primary brain cancer, and current treatments for glioblastoma are insufficient. HDAC6 is overexpressed in glioblastoma, and siRNA-mediated knockdown of HDAC6 inhibits glioma cell proliferation. Herein, we report a high-selective HDAC6 inhibitor, J22352, which
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