推荐产品
生物源
goat
品質等級
共軛
unconjugated
抗體表格
whole antiserum
抗體產品種類
primary antibodies
無性繁殖
polyclonal
包含
15 mM sodium azide
技術
indirect ELISA: 1:8,000
quantitative precipitin assay: 1.6-2.4 mg/mL
運輸包裝
dry ice
儲存溫度
−20°C
目標翻譯後修改
unmodified
一般說明
使用DNP-BSA作为免疫原在山羊体内产生了抗二硝基苯基（DNP）抗体。 抗血清已经过治疗，可去除脂蛋白。通过免疫电泳（IEP）确定该产品与DNP-BSA和DNP-RSA具有反应性，但与BSA（牛血清白蛋白）或RSA（兔血清白蛋白）无反应。
免疫原
DNP-BSA偶联物
應用
使用山羊生产的抗DNP抗体，通过时间分辨的磷光各向异性来研究IgE对FcεRI簇的影响。
山羊来源抗DNP抗体已用于DNA免疫FISH和胚泡的差异染色。
準備報告
脱脂
分析報告
通过免疫电泳和定量沉淀技术检测抗血清的特异性和效价。
免責聲明
除非我们的产品目录或产品附带的其他公司文档另有说明，否则我们的产品仅供研究使用，不得用于任何其他目的，包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。
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儲存類別代碼
10 - Combustible liquids
水污染物質分類（WGK）
nwg
閃點（°F）
Not applicable
閃點（°C）
Not applicable
其他客户在看
Miguel A Velazquez et al.
Biochimica et biophysica acta, 1864(2), 590-600 (2017-12-03)
Mouse maternal low protein diet exclusively during preimplantation development (Emb-LPD) is sufficient to programme altered growth and cardiovascular dysfunction in offspring. Here, we use an in vitro model comprising preimplantation culture in medium depleted in insulin and branched-chain amino acids
Jinming Song et al.
Biochemistry, 41(3), 881-889 (2002-01-16)
Rat mucosal-type mast cells of the RBL-2H3 line express a glycoprotein termed the MAst cell Function-associated Antigen (MAFA). When MAFA is clustered by its specific monoclonal antibody G63, secretion normally triggered by aggregating these cells' type I Fcepsilon receptor (FcepsilonRI)
M A Velazquez et al.
Human reproduction (Oxford, England), 31(9), 1970-1980 (2016-07-13)
Does advanced maternal age (AMA) in mice affect cardiometabolic health during post-natal life in offspring derived from an assisted reproduction technology (ART) procedure? Offspring derived from blastocysts collected from aged female mice displayed impaired body weight gain, blood pressure, glucose
Jinming Song et al.
Molecular immunology, 38(16-18), 1315-1321 (2002-09-10)
Clustering the mast cell function-associated antigen (MAFA), a membrane glycoprotein expressed on 2H3 cells, by its specific monoclonal antibody G63 substantially inhibits secretion normally triggered by aggregating these cells' Type I Fcepsilon receptor (FcepsilonRI). To explore possible MAFA-FcepsilonRI interactions giving
Shohreh Majd et al.
Current Alzheimer research, 15(8), 764-776 (2018-02-24)
Emerging evidence supports the hypothesis that metabolism dysfunction is involved in pathogenesis of Alzheimer's disease (AD). One aspect of metabolic dysfunction includes dysregulation of adenosine monophosphate kinase protein kinase (AMPK) and mammalian target of rapamycin (mTOR) metabolic axis, which is
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