推荐产品
生物源
mouse
品質等級
抗體表格
ascites fluid
抗體產品種類
primary antibodies
無性繁殖
monoclonal
物種活性(以同源性預測)
mammals
製造商/商標名
Chemicon®
技術
western blot: suitable
同型
IgG2a
NCBI登錄號
UniProt登錄號
運輸包裝
dry ice
目標翻譯後修改
unmodified
基因資訊
human ... TUBA1A(7846)
特異性
Alpha tubulin, detyrosinated.
免疫原
Epitope: detyrosinated
Synthetic peptide GEEEGE.
應用
(add note)
Research Category
Neuroscience
Neuroscience
Research Sub Category
Neurofilament & Neuron Metabolism
Neuronal & Glial Markers
Neurofilament & Neuron Metabolism
Neuronal & Glial Markers
This Anti-Tubulin Antibody, α, detyrosinated is validated for use in WB for the detection of Tubulin.
外觀
Liquid. Contains no preservative.
儲存和穩定性
Maintain at -20°C in undiluted aliquots for up to 6 months after date of receipt. Avoid repeated freeze/thaw cycles.
法律資訊
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
免責聲明
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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儲存類別代碼
10 - Combustible liquids
水污染物質分類(WGK)
WGK 1
閃點(°F)
Not applicable
閃點(°C)
Not applicable
Man to trypanosome: the tubulin tyrosination/detyrosination cycle revisited.
Cell motility and the cytoskeleton, 45(3), 173-184 (2000-03-08)
Cancer research, 61(13), 5024-5027 (2001-06-30)
Tubulin, the dimeric subunit of microtubules, is a major cell protein that is centrally involved in cell division. Tubulin is subject to specific enzymatic posttranslational modifications including cyclic tyrosine removal and addition at the COOH terminus of the alpha-subunit. Tubulin
Low-intensity pulsed ultrasound accelerates tooth movement via activation of the BMP-2 signaling pathway.
Testing null
The European journal of neuroscience, 34(3), 426-436 (2011-06-23)
Chronic stress causes various detrimental effects including cognitive and affective dysfunctions. Given the recent findings emphasizing the importance of information processing between the prefrontal cortex (PFC) and limbic structures on cognitive and affective functions, impairments of these functions caused by
Translational neurodegeneration, 4, 9-9 (2015-06-02)
In Alzheimer disease (AD), hyperphosphorylation of tau proteins results in microtubule destabilization and cytoskeletal abnormalities. Our prior ultra-morphometric studies documented a clear reduction in microtubules in pyramidal neurons in AD compared to controls, however, this reduction did not coincide with
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