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343226

Sigma-Aldrich

Nickel sulfide

99.7% trace metals basis, −150 mesh

Sinonimo/i:

Nickel subsulfide

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About This Item

Formula empirica (notazione di Hill):
Ni3S2
Numero CAS:
12035-72-2
Peso molecolare:
240.21
Numero CE:
234-829-6
Numero MDL:
MFCD00217044
Codice UNSPSC:
12161600
ID PubChem:
24861133
NACRES:
NA.23

Saggio

99.7% trace metals basis

Forma fisica

powder

Impiego in reazioni chimiche

reagent type: catalyst
core: nickel

Dimensione particelle

−150 mesh

Densità

5.8 g/mL at 25 °C (lit.)

applicazioni

battery manufacturing

Stringa SMILE

[Ni]=S=[Ni]=S=[Ni]

InChI

1S/3Ni.2S
YGHCWPXPAHSSNA-UHFFFAOYSA-N

Descrizione generale

Major phase is Ni3S2. Product may also include a small amount of sulfur-rich phase. Nickel Sulfide (Ni3S2) exists as a heazlewoodite mineral in nature. The structure of Ni3S2 is hombohedral. Raman spectral analysis of Ni3S2 has been reported.

Applicazioni

Nickel sulfide was implanted in mice, to induce the growth of malignant fibrous histiocytomas.

Confezionamento

Packaged in glass bottles

Avvertenze

Danger

Indicazioni di pericolo

H317,H331,H341,H350i,H372,H410

Classi di pericolo

Acute Tox. 3 Inhalation - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1A Inhalation - Muta. 2 - Skin Sens. 1 - STOT RE 1 Inhalation

Organi bersaglio

Lungs

Codice della classe di stoccaggio

6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects

Classe di pericolosità dell'acqua (WGK)

WGK 3

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable

Dispositivi di protezione individuale

Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges

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Certificati d'analisi (COA)

Lot/Batch Number
MKCP0417
MKCN2133
MKCF2714
MKCB8884
MKBX4005V

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Reactive oxygen-induced carcinogenesis causes hypermethylation of p16 (Ink4a) and activation of MAP kinase.
Govindarajan B, et al.
Molecular Medicine, 8(1) (2002)
C Huang et al.
Cancer research, 61(22), 8051-8057 (2001-11-24)
Nickel compounds induce cell transformation in cell culture models and tumor formation in experimental animals. However, the molecular mechanisms by which nickel compounds induce tumors are not yet well understood. The present study found that exposure of cells to either
Frank E Huggins et al.
Environmental science & technology, 45(14), 6188-6195 (2011-06-30)
XAFS spectroscopy has been used to determine the Ni species in particulate matter collected on quartz thimble filters in the stacks of eight residual (No. 6 fuel) oil-burning electric utility steam-generating units. Proper speciation of nickel in emitted particulate matter
Shosuke Kawanishi et al.
Environmental health perspectives, 110 Suppl 5, 789-791 (2002-11-12)
The U.S. National Toxicology Program has shown clear evidence of carcinogenicity of nickel subsulfide (Ni(3)S(2)) and some evidence of carcinogenicity of NiO (green) in rats. In the present study, DNA damage in cultured cells and in lungs of rats induced
A S Andrew et al.
American journal of physiology. Lung cellular and molecular physiology, 281(3), L607-L615 (2001-08-16)
Human epidemiological and animal studies have associated inhalation of nickel dusts with an increased incidence of pulmonary fibrosis. At the cellular level, particulate nickel subsulfide inhibits fibrinolysis by transcriptionally inducing expression of plasminogen activator inhibitor (PAI)-1, an inhibitor of the
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