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194751

Sigma-Aldrich

N-tert-Butylhydroxylamine hydrochloride

≥98%

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About This Item

Fórmula lineal:
(CH3)3CNHOH · HCl
Número de CAS:
57497-39-9
Peso molecular:
125.60
Beilstein/REAXYS Number:
3546053
EC Number:
260-771-6
MDL number:
MFCD00012598
UNSPSC Code:
12352100
PubChem Substance ID:
24851334
NACRES:
NA.22

assay

≥98%

form

solid

mp

183-185 °C (lit.)

SMILES string

Cl.CC(C)(C)NO

InChI

1S/C4H11NO.ClH/c1-4(2,3)5-6;/h5-6H,1-3H3;1H

InChI key

DCSATTBHEMKGIP-UHFFFAOYSA-N

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Application

N-tert-Butylhydroxylamine hydrochloride was used in spin trapping of short-lived radicals. It was also used in the synthesis of α-ketoamides and 3-spirocyclopropanated 2-azetidinones.

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves, type N95 (US)

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Yu-Kyung Kim et al.
Clinical hemorheology and microcirculation, 40(4), 315-324 (2009-01-08)
Irradiation has been shown to induce biochemical changes in stored red blood cells (RBCs) and to generate reactive oxygen species (ROS). This study evaluated the hemorheological properties, the degree of lipid peroxidation and the oxidative susceptibility of irradiated RBCs. Furthermore
Hyun Jeong Kim et al.
Redox report : communications in free radical research, 10(6), 287-293 (2006-01-28)
Heat shock may increase oxidative stress due to increased production of reactive oxygen species and/or the promotion of cellular oxidation events. Therefore, compounds that scavenge reactive oxygen species may regulate heat shock-induced cell death. Recently, it has been shown that
Jin Hyup Lee et al.
Carcinogenesis, 25(8), 1435-1442 (2004-03-16)
Exposure of cells to ionizing radiation leads to formation of reactive oxygen species (ROS) that are associated with radiation-induced cytotoxicity. Therefore, compounds that scavenge ROS may confer radioprotective effects. Recently, it has been shown that the decomposition product of the
David W Killilea et al.
Antioxidants & redox signaling, 5(5), 507-516 (2003-10-29)
Iron accumulates as a function of age in several tissues in vivo and is associated with the pathology of numerous age-related diseases. The molecular basis of this change may be due to a loss of iron homeostasis at the cellular
On the anti-aging activities of aminoguanidine and N-t-butylhydroxylamine.
A R Hipkiss
Mechanisms of ageing and development, 122(2), 169-171 (2001-02-13)
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