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Key Documents

3951

Sigma-Aldrich

高碘酸 溶液

1 g/dL in deionized water

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About This Item

MDL號碼:
分類程式碼代碼:
12352106
PubChem物質ID:
NACRES:
NA.47

形狀

solution

儲存期限

expiry date on label

反應適用性

reagent type: oxidant

IVD

for in vitro diagnostic use

濃度

1 g/dL in deionized water

pH值

1.77

應用

hematology
histology

運輸包裝

wet ice

儲存溫度

2-8°C

SMILES 字串

OI(O)(O)(O)(O)=O

InChI

1S/H5IO6/c2-1(3,4,5,6)7/h(H5,2,3,4,5,6,7)

InChI 密鑰

TWLXDPFBEPBAQB-UHFFFAOYSA-N

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應用

For use in procedure no. 395 for Periodic Acid-Schiff (PAS) and Silver Stain Procedure No. HT100.

象形圖

Corrosion

訊號詞

Danger

危險聲明

危險分類

Aquatic Chronic 3 - Eye Dam. 1 - Skin Corr. 1

儲存類別代碼

8B - Non-combustible corrosive hazardous materials

水污染物質分類(WGK)

WGK 2

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter


分析證明 (COA)

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Globoid cell leukodystrophy (GLD), or Krabbe disease, is an inherited, neurologic disorder that results from deficiency of a lysosomal enzyme, galactosylceramidase. Most commonly, deficits of galactosylceramidase result in widespread central and peripheral nervous system demyelination and death in affected infants
Sonja Frimmel et al.
Journal of ophthalmic & vision research, 12(2), 175-182 (2017-05-26)
Diabetic retinopathy is a leading cause of vision loss. There is a great need for early diagnosis prior to the occurrence of irreversible structural damages. Expression of endothelial adhesion molecules is observed before the onset of diabetic vascular damage; however
Allison M Bradbury et al.
Journal of neuroscience research, 94(11), 1007-1017 (2016-09-18)
Globoid cell leukodystrophy (GLD), or Krabbe's disease, is a debilitating and always fatal pediatric neurodegenerative disease caused by a mutation in the gene encoding the hydrolytic enzyme galactosylceramidase (GALC). In the absence of GALC, progressive loss of myelin and accumulation
Daniela Rovito et al.
Nucleic acids research, 49(8), 4472-4492 (2021-04-10)
Skeletal muscle is a dynamic tissue the size of which can be remodeled through the concerted actions of various cues. Here, we investigated the skeletal muscle transcriptional program and identified key tissue-specific regulatory genetic elements. Our results show that Myod1
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Successful treatment of HIV-infected patients with combinational antiretroviral therapies (cART) can now prolong patients' lives to nearly normal life spans. However, the new challenge faced by many of those HIV-infected patients is chronic neuroinflammation and neurotoxicity that often leads to

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