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Merck
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重要文件

F5879

Sigma-Aldrich

柠檬酸铁 (III) 铵

reagent grade, powder

同義詞:

枸橼酸铁铵, 柠檬酸胺铁

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About This Item

線性公式:
C6H8O7 ·xFe3+ · yNH3
CAS號碼:
EC號碼:
分類程式碼代碼:
12352300
eCl@ss:
38190508
PubChem物質ID:
NACRES:
NA.55

等級

reagent grade

品質等級

形狀

powder

成份

Iron, 16.5-18.5%

正離子痕跡

Fe: 16.5-18.5%

SMILES 字串

N.OC12CC(=O)O[Fe](OC(=O)C1)OC(=O)C2

InChI

1S/C6H8O7.Fe.H3N/c7-3(8)1-6(13,5(11)12)2-4(9)10;;/h13H,1-2H2,(H,7,8)(H,9,10)(H,11,12);;1H3/q;+3;/p-3

InChI 密鑰

FRHBOQMZUOWXQL-UHFFFAOYSA-K

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一般說明

柠檬酸铁铵(III)可作为试剂和铁前体用于多种有机反应和 Fe3O4 基纳米材料的制备。用柠檬酸和 NH4OH 按不同比例处理 Fe(OH)3,可制备。

應用

柠檬酸铁铵可作为试剂,在 HEPES (4-(2-羟乙基)-1-哌嗪乙磺酸)缓冲液环境下,从含铁细胞制备两亲性铁载体含铁细胞络合物。它还可以作为铁前体制备 Fe3O4-碳点磁荧光杂化纳米颗粒。

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 1

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Pengfei Wang et al.
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The in situ measurement of the distribution of biomolecules inside a cell is one of the important goals in life science. Among various imaging techniques, magnetic imaging (MI) based on the nitrogen-vacancy (NV) center in diamond provides a powerful tool
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Nature communications, 8(1), 403-403 (2017-09-03)
Hepcidin regulates systemic iron homeostasis. Suppression of hepcidin expression occurs physiologically in iron deficiency and increased erythropoiesis but is pathologic in thalassemia and hemochromatosis. Here we show that epigenetic events govern hepcidin expression. Erythropoiesis and iron deficiency suppress hepcidin via
Yinghui Zhang et al.
Antioxidants & redox signaling, 13(7), 999-1009 (2010-04-22)
Cytosolic ferritins sequester and store iron, consequently protecting cells against iron-mediated free radical damage. However, the mechanisms of iron exit from the ferritin cage and reutilization are largely unknown. In a previous study, we found that mitochondrial ferritin (MtFt) expression
Rita Polati et al.
Journal of proteomics, 76 Spec No., 10-27 (2012-07-28)
Macrophages play a critical role at the crossroad between iron metabolism and immunity, being able to store and recycle iron derived from the phagocytosis of senescent erythrocytes. The way by which macrophages manage non-heme iron at physiological concentration is still
Yulia Pollak et al.
Journal of neural transmission (Vienna, Austria : 1996), 120(1), 37-48 (2012-03-27)
Iron accumulation and iron-related oxidative stress are involved in several pathological conditions and provide a rationale for the development of iron chelators as novel promising therapeutic strategies. Thus, we have recently synthesized multifunctional non-toxic, brain permeable iron chelating compounds, M30

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