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Merck

L3140

Sigma-Aldrich

脂磷壁酸 来源于化脓链球菌

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About This Item

CAS号:
MDL號碼:
分類程式碼代碼:
12352211
NACRES:
NA.25

生物源

Streptococcus pyogenes

品質等級

形狀

lyophilized powder

官能基

phospholipid

脂質類型

polymerizable lipids

運輸包裝

ambient

儲存溫度

2-8°C

應用

脂磷壁酸 (LTA) 是革兰氏阳性菌细胞壁的一种复杂成分,参与广泛的细胞过程,如刺激免疫应答和细胞信号通路。LTA 在革兰氏阳性菌物种之间存在差异。 化脓性链球菌 产生的脂磷壁酸可用于比较其结构、免疫原性和与其他细菌 LTA 的功能。

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

Eyeshields, Gloves, type N95 (US)


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J P Himanen et al.
Journal of general microbiology, 139(11), 2659-2665 (1993-11-01)
To evaluate the suitability of Bacillus subtilis as a production host of heterologous proteins for pharmaceutical purposes, we assessed the biological activity of this bacterium and its major cell envelope components, lipoteichoic acid (LTA) and peptidoglycan-teichoic acid complex (PG-TA) in
Ning Xie et al.
Bulletin du cancer, 99(5), E55-E63 (2012-04-24)
To investigate the therapeutic efficacy of lipoteichoic acid of Bifidobacterium (BLTA) in combination with 5-fluorouracil (5-FU) treatment on the mice bearing inoculated hepatoma-22 (H(22)) cells and the effects of BLTA on immunological regulation of organism, and explore its mechanisms. Tumor-bearing
Induction of intestinal pro-inflammatory immune responses by lipoteichoic acid.
Zadeh M, Khan MW, Goh YJ, et al.
Journal of Inflammation, 9, 7-7 (2012)
Jun Ho Jeon et al.
Comparative immunology, microbiology and infectious diseases, 35(4), 363-374 (2012-03-27)
At allergic inflammation, cross-linking of FcɛRI with multivalent antigen-bound IgE triggers the signaling pathways via activation of protein kinase C (PKC) and mobilization of intracellular Ca(2+) leading to the production of various mediators such as interleukin-6 (IL-6). Accumulating reports demonstrated
Hun Yi Park et al.
International journal of pediatric otorhinolaryngology, 76(4), 475-479 (2012-02-07)
Change in matrix metalloproteinases (MMPs) and regulation of their tissue inhibitors of metalloproteinases (TIMPs) could play certain role in the pathogenesis of otitis media. This study was designed to evaluate the modulation of MMPs and TIMPs in middle ear epithelium

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