推荐产品
等級
SAJ special grade
蒸汽密度
2.69 (vs air)
蒸汽壓力
1 mmHg ( 20 °C)
化驗
≥99.0%
形狀
liquid
expl. lim.
18 %
存貨情形
available only in Japan
濃度
14.3 M (pure liquid)
dilution
(for analytical testing)
折射率
n20/D 1.500 (lit.)
pH值
4.5-6 (20 °C, 500 g/L)
bp
157 °C (lit.)
密度
1.114 g/mL at 25 °C (lit.)
SMILES 字串
OCCS
InChI
1S/C2H6OS/c3-1-2-4/h3-4H,1-2H2
InChI 密鑰
DGVVWUTYPXICAM-UHFFFAOYSA-N
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相关类别
訊號詞
Danger
危險分類
Acute Tox. 2 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Acute 1 - Aquatic Chronic 2 - Eye Dam. 1 - Repr. 2 - Skin Irrit. 2 - Skin Sens. 1A - STOT RE 2 Oral
標靶器官
Liver,Heart
儲存類別代碼
6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials
水污染物質分類（WGK）
WGK 3
閃點（°F）
165.2 °F - closed cup
閃點（°C）
74 °C - closed cup
個人防護裝備
Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter
Leon Tribolet et al.
The Journal of infectious diseases, 211(3), 416-425 (2014-08-21)
Na-ASP-2 is an efficacious hookworm vaccine antigen. However, despite elucidation of its crystal structure and studies addressing its immunobiology, the function of Na-ASP-2 has remained elusive. We probed a 9000-protein human proteome microarray with Na-ASP-2 and showed binding to CD79A
Katharina Rothe et al.
Blood, 123(23), 3622-3634 (2014-04-24)
Previous studies demonstrated that imatinib mesylate (IM) induces autophagy in chronic myeloid leukemia (CML) and that this process is critical to cell survival upon therapy. However, it is not known if the autophagic process differs at basal levels between CML
E M Michalak et al.
Cell death and differentiation, 15(6), 1019-1029 (2008-02-09)
The ability of p53 to induce apoptosis in cells with damaged DNA is thought to contribute greatly to its tumour suppressor function. P53 has been proposed to induce apoptosis via numerous transcriptional targets or even by direct cytoplasmic action. Two
Nobuko Akiyama et al.
The Journal of experimental medicine, 211(12), 2425-2438 (2014-11-12)
Medullary thymic epithelial cells (mTECs) expressing the autoimmune regulator AIRE and various tissue-specific antigens (TSAs) are critical for preventing the onset of autoimmunity and may attenuate tumor immunity. However, molecular mechanisms controlling mTEC development remain elusive. Here, we describe the
Yoh Arita et al.
Nature communications, 5, 4552-4552 (2014-07-30)
The origin and developmental mechanisms underlying coronary vessels are not fully elucidated. Here we show that myocardium-derived angiopoietin-1 (Ang1) is essential for coronary vein formation in the developing heart. Cardiomyocyte-specific Ang1 deletion results in defective formation of the subepicardial coronary
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