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T2449

Sigma-Aldrich

Triglyceride Reagent

used for quantitative enzymatic determination of triglyerides

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About This Item

UNSPSC Code:
12352200
NACRES:
NA.25

usage

10 mL sufficient for 50 tests

storage temp.

2-8°C

Application

The triglyceride and free glycerol kits are for the quantitative determination of glycerol, total triglycerides or free triglycerides.

Principle

The triglyceride reagent includes lipase for hydrolysis of triglycerides to glycerol.

Linkage

In addition to kits, the individual reagents and glycerol standard are available separately when fewer reactions are needed.

related product

Product No.
Description
Pricing

required but not provided

standard

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves, type N95 (US)


Certificates of Analysis (COA)

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Hideaki Miyoshi et al.
Journal of cellular biochemistry, 105(6), 1430-1436 (2008-11-05)
In adipocytes, lipid droplet (LD) size reflects a balance of triglyceride synthesis (lipogenesis) and hydrolysis (lipolysis). Perilipin A (Peri A) is the most abundant phosphoprotein on the surface of adipocyte LDs and has a crucial role in lipid storage and
Haoyong Yu et al.
Journal of molecular endocrinology, 59(2), 121-128 (2017-06-02)
5-Aminolevulinic acid (5-ALA), the first compound in the porphyrin synthesis pathway, has been reported to ameliorate the diabetic state in Otsuka Long-Evans Tokushima Fatty rats by reducing fat pad weight in the retroperitoneal region. Dietary supplementation with 5-ALA has additionally
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Diabetes, 58(6), 1391-1402 (2009-03-28)
Glycogen synthase kinase (GSK)-3beta plays an important role in cardiomyopathies. Cardiac-specific metallothionein-overexpressing transgenic (MT-TG) mice were highly resistant to diabetes-induced cardiomyopathy. Therefore, we investigated whether metallothionein cardiac protection against diabetes is mediated by inactivation of GSK-3beta. Diabetes was induced with
Ana Guarner et al.
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To understand the consequences of the complete elimination of E2F regulation, we profiled the proteome of Drosophila dDP mutants that lack functional E2F/DP complexes. The results uncovered changes in the larval fat body, a differentiated tissue that grows via endocycles.
Hila Korach-Rechtman et al.
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