AP504P
Goat Anti-Human IgG γ chain Antibody, HRP conjugate, Species Adsorbed
Chemicon®, from goat
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About This Item
UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.46
Recommended Products
biological source
goat
Quality Level
conjugate
peroxidase conjugate
antibody form
affinity purified immunoglobulin
antibody product type
secondary antibodies
clone
polyclonal
species reactivity
human
manufacturer/tradename
Chemicon®
technique(s)
ELISA: suitable
shipped in
wet ice
target post-translational modification
unmodified
Related Categories
General description
IgG is one of the most abundant protein in human serum. IgG is important for our defence against microorganisms and the molecules are produced by B-lymphocytes as a part of our adaptive immune response. The IgG molecule has two separate functions; to bind to the pathogen that elicited the response and to recruit other cells and molecules to destroy the antigen. The variability of the IgG pool is generated by somatic recombination.
Specificity
Reacts with with the heavy chain of human IgG. Absorbed for human IgM, IgD and IgA.
Application
Detect Human IgG γ chain using this Goat anti-Human IgG γ chain Antibody, HRP conjugate, Species Adsorbed validated for use in ELISA.
Physical form
Purified by affinity chromatography on pooled human IgG covalently linked to agarose. Liquid in 50% glycerol/50% PBS, pH 7.4. No preservative added.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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Storage Class
10 - Combustible liquids
wgk_germany
WGK 2
Certificates of Analysis (COA)
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Lisa M Smith et al.
Journal of virology, 95(23), e0088221-e0088221 (2021-09-23)
Human and simian immunodeficiency virus (HIV and SIV) infections establish lifelong reservoirs of cells harboring an integrated proviral genome. Genome editing CRISPR-associated Cas9 nucleases, combined with SIV-specific guiding RNA (gRNA) molecules, inactivate integrated provirus DNA in vitro and in animal
Adam L Bailey et al.
bioRxiv : the preprint server for biology (2020-11-12)
Epidemiological studies of the COVID-19 pandemic have revealed evidence of cardiac involvement and documented that myocardial injury and myocarditis are predictors of poor outcomes. Nonetheless, little is understood regarding SARS-CoV-2 tropism within the heart and whether cardiac complications result directly
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