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T2449

Sigma-Aldrich

Triglyceride Reagent

used for quantitative enzymatic determination of triglyerides

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About This Item

Kod UNSPSC:
12352200
NACRES:
NA.25

zastosowanie

10 mL sufficient for 50 tests

temp. przechowywania

2-8°C

Zastosowanie

The triglyceride and free glycerol kits are for the quantitative determination of glycerol, total triglycerides or free triglycerides.

Zasada

The triglyceride reagent includes lipase for hydrolysis of triglycerides to glycerol.

Powiązanie

In addition to kits, the individual reagents and glycerol standard are available separately when fewer reactions are needed.
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produkt powiązany

Numer produktu
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08168

Timestrip Plus™ 8 °C

standardowe

Numer produktu
Opis
Cennik

wymagane, ale niedostarczone

Numer produktu
Opis
Cennik

Kod klasy składowania

11 - Combustible Solids

Klasa zagrożenia wodnego (WGK)

WGK 3

Temperatura zapłonu (°F)

Not applicable

Temperatura zapłonu (°C)

Not applicable

Środki ochrony indywidualnej

Eyeshields, Gloves, type N95 (US)

Certyfikaty analizy (CoA)

Poszukaj Certyfikaty analizy (CoA), wpisując numer partii/serii produktów. Numery serii i partii można znaleźć na etykiecie produktu po słowach „seria” lub „partia”.

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Journal of cellular biochemistry, 105(6), 1430-1436 (2008-11-05)
In adipocytes, lipid droplet (LD) size reflects a balance of triglyceride synthesis (lipogenesis) and hydrolysis (lipolysis). Perilipin A (Peri A) is the most abundant phosphoprotein on the surface of adipocyte LDs and has a crucial role in lipid storage and
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Yuehui Wang et al.
Diabetes, 58(6), 1391-1402 (2009-03-28)
Glycogen synthase kinase (GSK)-3beta plays an important role in cardiomyopathies. Cardiac-specific metallothionein-overexpressing transgenic (MT-TG) mice were highly resistant to diabetes-induced cardiomyopathy. Therefore, we investigated whether metallothionein cardiac protection against diabetes is mediated by inactivation of GSK-3beta. Diabetes was induced with
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Developmental cell, 43(6), 689-703 (2017-12-14)
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Low-fat diets and exercise are generally assumed to ameliorate obesity-related metabolic dysfunctions, but the importance of exercise vs. dietary changes is debated. Male C57BL/6J mice were fed a high-fat/high-sucrose (HF/HS) diet to induce obesity and then either maintained on the
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