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Merck
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Key Documents

209A-1

Sigma-Aldrich

LH Rabbit Polyclonal Antibody

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Wszystkie zdjęcia(1)

About This Item

Kod UNSPSC:
12352203
NACRES:
NA.41

pochodzenie biologiczne

rabbit

Poziom jakości

100
500

białko sprzężone

unconjugated

forma przeciwciała

Ig fraction of antiserum

rodzaj przeciwciała

primary antibodies

klon

polyclonal

opis

For In Vitro Diagnostic Use in Select Regions (See Chart)

Postać

buffered aqueous solution

reaktywność gatunkowa

human

opakowanie

vial of 0.1 mL concentrate (209A-14)
vial of 0.5 mL concentrate (209A-15)
bottle of 1.0 mL predilute (209A-17)
vial of 1.0 mL concentrate (209A-16)
bottle of 7.0 mL predilute (209A-18)

producent / nazwa handlowa

Cell Marque™

metody

immunohistochemistry (formalin-fixed, paraffin-embedded sections): 1:100-1:500

kontrola

pituitary

Warunki transportu

wet ice

temp. przechowywania

2-8°C

wizualizacja

cytoplasmic

Opis ogólny

Anti-LH is a useful marker in classification of pituitary tumors and the study of pituitary disease. It reacts with LH-producing cells (gonadotrophs).

Jakość


IVD
IVD
IVD
RUO

Powiązanie

LH Positive Control Slides, Product No. 209S, are available for immunohistochemistry (formalin-fixed, paraffin-embedded sections).

Postać fizyczna

Solution in Tris Buffer, pH 7.3-7.7, with 1% BSA and <0.1% Sodium Azide

Uwaga dotycząca przygotowania

Download the IFU specific to your product lot and formatNote: This requires a keycode which can be found on your packaging or product label.

Inne uwagi

For Technical Service please contact: 800-665-7284 or email: service@cellmarque.com

Informacje prawne

Cell Marque is a trademark of Merck KGaA, Darmstadt, Germany
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Masz już ten produkt?

Dokumenty związane z niedawno zakupionymi produktami zostały zamieszczone w Bibliotece dokumentów.

Odwiedź Bibliotekę dokumentów

K Saccomanno et al.
The Journal of clinical endocrinology and metabolism, 78(5), 1103-1107 (1994-05-01)
Human nonfunctioning pituitary adenomas (NFPA) may produce CG in addition to the classical glycoprotein hormones (LH, FSH, and TSH). The aim of the present study was to localize LH beta, FSH beta, TSH beta, alpha-subunit (alpha SU), CG, and its
T Sano et al.
Virchows Archiv. A, Pathological anatomy and histopathology, 417(4), 361-367 (1990-01-01)
Two pituitary adenomas removed from a 37-year-old woman and a 26-year-old woman with typical Cushing's disease were studied by light and electron microscopy, immunohistochemistry and radioimmunoassay of tissue culture media. Both patients had high plasma levels of cortisol and normal
J J Kovalic et al.
Journal of neuro-oncology, 16(3), 227-232 (1993-06-01)
There is general agreement that postoperative radiation therapy is beneficial for patients with subtotally resected pituitary adenomas. We have identified 41 such patients treated during a 20-year period who received postoperative irradiation for a pituitary adenoma. The usual dose was
I Felix et al.
Human pathology, 22(7), 719-721 (1991-07-01)
A 19-year-old man with blurred vision, headache, and no signs or symptoms of hormone excess was found to have a pituitary adenoma. The tumor was removed by a transfrontal approach. He had postoperative radiation therapy, but subsequently had three recurrences
S La Rosa et al.
Virchows Archiv : an international journal of pathology, 437(3), 264-269 (2000-10-19)
Gonadotropin-releasing hormone (GnRH), which is a well-known regulator of gonadotroph function, has recently been considered to be a paracrine factor involved in the control of somatotroph, lactotroph, and corticotroph cells. GnRH action is initiated by binding to a specific cell

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