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Merck
모든 사진(1)

주요 문서

O5003

Sigma-Aldrich

OPI Media Supplement

Hybri-Max, γ-irradiated, powder, suitable for hybridoma

동의어(들):

OPI Media for cell culture

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About This Item

UNSPSC 코드:
12352207
NACRES:
NA.75

Grade

Hybri-Max

Quality Level

무균

γ-irradiated

형태

powder

농도

18-40 mg/vial

기술

cell culture | hybridoma: suitable

불순물

endotoxin, tested

저장 온도

−20°C

애플리케이션

OPI Media Supplement has been used to grow hybridoma cell lines from XenoMouse strains and BALB/c female mice.

기타 정보

Formulated with: 0.15 g oxaloacetate, 0.05 g pyruvate, and 0.0082 g insulin.

재구성

Reconstitute contents of vial with 10 mL sterile water. Do not use cell culture medium for initial reconstitution. Each vial sufficient to prepare 1 liter of medium. Final working concentration: 1 mM oxaloacetate, 0.45 mM pyruvate, 0.2 U/ml insulin.

법적 정보

Hybri-Max is a trademark of Sigma-Aldrich Co. LLC

픽토그램

CorrosionExclamation mark

신호어

Danger

유해 및 위험 성명서

Hazard Classifications

Eye Dam. 1 - Skin Corr. 1 - Skin Sens. 1

Storage Class Code

8A - Combustible corrosive hazardous materials

WGK

WGK 1

Flash Point (°F)

Not applicable

Flash Point (°C)

Not applicable


시험 성적서(COA)

제품의 로트/배치 번호를 입력하여 시험 성적서(COA)을 검색하십시오. 로트 및 배치 번호는 제품 라벨에 있는 ‘로트’ 또는 ‘배치’라는 용어 뒤에서 찾을 수 있습니다.

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문서 라이브러리 방문

Andrew W Drake et al.
Analytical biochemistry, 328(1), 35-43 (2004-04-15)
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Tzu-Ling Sung et al.
PLoS pathogens, 5(1), e1000263-e1000263 (2009-01-17)
Cyclin T1 is a regulatory subunit of a general RNA polymerase II elongation factor known as P-TEFb. Cyclin T1 is also required for Tat transactivation of HIV-1 LTR-directed gene expression. Translation of Cyclin T1 mRNA has been shown to be
Megan E Bosch et al.
Journal of neurochemistry, 148(5), 612-624 (2018-07-03)
Juvenile neuronal ceroid lipofuscinosis (JNCL) is a lysosomal storage disease caused by autosomal recessive mutations in ceroid lipofuscinosis 3 (CLN3). Children with JNCL experience progressive visual, cognitive, and motor deterioration with a decreased life expectancy (late teens-early 20s). Neuronal loss

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