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Merck
모든 사진(1)

주요 문서

AP130F

Sigma-Aldrich

Goat Anti-Mouse IgG & IgM Antibody, FITC conjugate

Chemicon®, from goat

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About This Item

UNSPSC 코드:
12352203
eCl@ss:
32160702
NACRES:
NA.46

생물학적 소스

goat

Quality Level

결합

FITC conjugate

항체 형태

F(ab′)2 fragment of affinity isolated antibody

항체 생산 유형

secondary antibodies

클론

polyclonal

종 반응성

mouse

제조업체/상표

Chemicon®

기술

immunofluorescence: suitable

배송 상태

wet ice

타겟 번역 후 변형

unmodified

애플리케이션

Detect Mouse IgG & IgM using this Goat anti-Mouse IgG & IgM Antibody, FITC conjugate validated for use in IF.
Research Category
Secondary & Control Antibodies
Research Sub Category
Whole Immunoglobulin Secondary Antibodies

법적 정보

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

면책조항

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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유해 및 위험 성명서

예방조치 성명서

Hazard Classifications

Aquatic Chronic 3

Storage Class Code

11 - Combustible Solids

WGK

WGK 3


시험 성적서(COA)

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문서 라이브러리 방문

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Experimental and therapeutic medicine, 23(4), 273-273 (2022-03-08)
Osteoporosis (OP) is a systemic metabolic bone disease that occurs most frequently in the elderly. The main pathogenesis of OP is excessive proliferation and differentiation of osteoclasts, in which the peroxisome proliferator-activated receptor γ (PPARγ) pathway has a pivotal role.
Di Xie et al.
Journal of neuroinflammation, 17(1), 57-57 (2020-02-18)
Microglia activation is associated with the development of hypoxic-ischemic brain injury (HIBI). Neuroinflammation suppression might be a suitable therapeutic target in hypoxic oligodendrocyte injury. This study aims to determine whether clemastine can improve hypomyelination by suppressing the activated microglia and
Yiyu Deng et al.
PloS one, 9(1), e87420-e87420 (2014-02-06)
Hypoxic exposure in the perinatal period causes periventricular white matter damage (PWMD), a condition associated with myelination abnormalities. Under hypoxic conditions, glial cells were activated and released a large number of inflammatory mediators in the PWM in neonatal brain, which

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