524632
Phosphatase Inhibitor Cocktail V
50X, lyophilized solid, for the inhibition of acid, alkaline, serine/threonine and protein tyrosine phosphatases. This small molecule/inhibitor is primarily used for Phosphorylation & Dephosphorylation applications.
동의어(들):
Phosphatase inhibitor cocktail
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모든 사진(1)
About This Item
추천 제품
product name
Phosphatase Inhibitor Cocktail Set V, 50X, Lyophilized, The Phosphatase Inhibitor Cocktail Set V, 50X, Lyophilized controls the activity of Phosphatase. This small molecule/inhibitor is primarily used for Phosphorylation & Dephosphorylation applications.
형태
lyophilized solid
제조업체/상표
Calbiochem®
저장 조건
OK to freeze
desiccated (hygroscopic)
protect from light
배송 상태
ambient
저장 온도
2-8°C
일반 설명
A cocktail of four phosphatase inhibitors for broad-spectrum inhibition of acid, alkaline, serine/threonine and protein tyrosine phosphatases. Available as a 1 ml vial or a set of five 1 ml vials. Each vial, when reconstituted with 1 ml H2O contains 250 mM Sodium Fluoride, 50 mM β-Glycerophosphate (Cat. No. 35675), 50 mM Sodium Pyrophosphate Decahydrate, 50 mM Sodium Orthovanadate.
A cocktail of four phosphatase inhibitors for broad-spectrum inhibition of acid, alkaline, serine/threonine and protein tyrosine phosphatases. Available as a 1 ml vial or a set of five 1 ml vials. Once reconstituted, each vial contains the following components:
포장
Packaged under inert gas
경고
Toxicity: Toxic (F)
재구성
Following reconstitution, aliquot and freeze (-20°C) for long-term storage. Dilute 1:50 just prior to use.
Reconstitute each vial with 1 ml H₂O.
법적 정보
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
신호어
Danger
유해 및 위험 성명서
Hazard Classifications
Acute Tox. 4 Oral - Eye Dam. 1 - Skin Irrit. 2
보충제 위험성
Storage Class Code
11 - Combustible Solids
WGK
WGK 3
Flash Point (°F)
Not applicable
Flash Point (°C)
Not applicable
시험 성적서(COA)
제품의 로트/배치 번호를 입력하여 시험 성적서(COA)을 검색하십시오. 로트 및 배치 번호는 제품 라벨에 있는 ‘로트’ 또는 ‘배치’라는 용어 뒤에서 찾을 수 있습니다.
International journal of molecular sciences, 23(13) (2022-07-10)
An oversupply of nutrients with a loss of metabolic flexibility and subsequent cardiac dysfunction are hallmarks of diabetic cardiomyopathy. Even if excess substrate is offered, the heart suffers energy depletion as metabolic fluxes are diminished. To study the effects of
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