모든 사진(3)

주요 문서

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A7205

3-Acetamidophenol

Name: 3-Acetamidophenol
Brand: ALDRICH

97%

동의어(들):

3′-Hydroxyacetanilide

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모든 사진(3)

About This Item

Linear Formula:

CH₃CONHC₆H₄OH

CAS Number:

621-42-1

Molecular Weight:

151.16

Beilstein:

907998

EC Number:

210-687-0

MDL number:

MFCD00002263

UNSPSC 코드:

12352100

PubChem Substance ID:

24891188

NACRES:

NA.22

추천 제품

Slide 1 of 1

1 of 1

Sigma-Aldrich

Q3625

Quinidine

Quality Level

200

분석

97%

형태

crystals

mp

145-148 °C (lit.)

SMILES string

CC(=O)Nc1cccc(O)c1

InChI

1S/C8H9NO2/c1-6(10)9-7-3-2-4-8(11)5-7/h2-5,11H,1H3,(H,9,10)

InChI key

QLNWXBAGRTUKKI-UHFFFAOYSA-N

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픽토그램

GHS07

신호어

Warning

유해 및 위험 성명서

H315,H319

예방조치 성명서

P264 - P280 - P302 + P352 - P305 + P351 + P338 - P332 + P313 - P337 + P313

Hazard Classifications

Eye Irrit. 2 - Skin Irrit. 2

Storage Class Code

11 - Combustible Solids

WGK

WGK 2

Flash Point (°F)

Not applicable

Flash Point (°C)

Not applicable

개인 보호 장비

dust mask type N95 (US), Eyeshields, Gloves

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문서 라이브러리 방문

Identification of hepatic protein targets of the reactive metabolites of the non-hepatotoxic regioisomer of acetaminophen, 3'-hydroxyacetanilide, in the mouse in vivo using two-dimensional gel electrophoresis and mass spectrometry.

Y Qiu et al.

Advances in experimental medicine and biology, 500, 663-673 (2002-01-05)

Acetaminophen induces apoptosis of C6 glioma cells by activating the c-Jun NH(2)-terminal protein kinase-related cell death pathway.

M A Bae et al.

Molecular pharmacology, 60(4), 847-856 (2001-09-20)

Acetaminophen (AAP), a widely used analgesic drug, can damage various organs when taken in large doses. In this study, we investigate whether AAP causes cell damage by altering the early signaling pathways associated with cell death and survival. AAP caused

Subcellular binding and effects on calcium homeostasis produced by acetaminophen and a nonhepatotoxic regioisomer, 3'-hydroxyacetanilide, in mouse liver.

M A Tirmenstein et al.

The Journal of biological chemistry, 264(17), 9814-9819 (1989-06-15)

Acetaminophen (250 mg/kg) administered intraperitoneally to fasted, phenobarbital-induced mice produced hepatotoxicity. No hepatotoxicity was observed after the administration of the regioisomer 3'-hydroxyacetanilide (600 mg/kg). Similar levels of covalent binding to liver homogenates occurred in mice receiving either acetaminophen or 3'-hydroxyacetanilide

Glutathione depletion in human erythrocytes as an indicator for microsomal activation of cyclophosphamide and 3-hydroxyacetanilide.

N G Palmen et al.

Toxicology, 84(1-3), 157-170 (1993-11-12)

A model system for the detection of reactive metabolites, using glutathione depletion after microsomal activation, has been described previously. We developed a battery of complementary test systems using rat liver microsomes for metabolism and aqueous glutathione solutions, human erythrocytes or

A comparative study of mouse liver proteins arylated by reactive metabolites of acetaminophen and its nonhepatotoxic regioisomer, 3'-hydroxyacetanilide.

T G Myers et al.

Chemical research in toxicology, 8(3), 403-413 (1995-04-01)

Acetaminophen (4'-hydroxyacetanilide), a widely used analgesic/antipyretic drug, is hepatotoxic in large doses, whereas the m-hydroxy isomer of acetaminophen, 3'-hydroxyacetanilide, is not hepatotoxic. Both are oxidized by mouse liver cytochromes P-450 to reactive metabolites that bind covalently to hepatic proteins. Because

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