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Magnetic Stir Bar

for use with For 3.0-5.0mL vessels, pkg of 6 ea, PTFE

Sinonimo/i:

STIRRING BARS 3ML TO 5ML M.V. PK6

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About This Item

Codice UNSPSC:
41103806

Materiali

PTFE
triangular PTFE-coated

Confezionamento

pkg of 6 ea

Compatibilità

for use with For 3.0-5.0mL vessels

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Note legali

WHEATON is a registered trademark of Wheaton Industries, Inc.

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Shuo Liu et al.
Cell, 176(3), 491-504 (2019-01-08)
Increased protein synthesis plays an etiologic role in diverse cancers. Here, we demonstrate that METTL13 (methyltransferase-like 13) dimethylation of eEF1A (eukaryotic elongation factor 1A) lysine 55 (eEF1AK55me2) is utilized by Ras-driven cancers to increase translational output and promote tumorigenesis in vivo.
Ricardo Usategui-Martín et al.
Molecular therapy. Methods & clinical development, 17, 1155-1166 (2020-06-10)
Retinal photoreceptor degeneration occurs frequently in several neurodegenerative retinal diseases such as age-related macular degeneration, retinitis pigmentosa, or genetic retinal diseases related to the photoreceptors. Despite the impact on daily life and the social and economic consequences, there is no
Hyo Min Cho et al.
Molecular cell, 73(2), 364-376 (2018-12-26)
Mitophagy, a mitochondrial quality control process for eliminating dysfunctional mitochondria, can be induced by a response of dynamin-related protein 1 (Drp1) to a reduction in mitochondrial membrane potential (MMP) and mitochondrial division. However, the coordination between MMP and mitochondrial division
Jonathan Martinez-Fabregas et al.
Cell reports, 33(12), 108545-108545 (2020-12-29)
Cytokines are highly pleiotropic ligands that regulate the immune response. Here, using interleukin-6 (IL-6) as a model system, we perform detailed phosphoproteomic and transcriptomic studies in human CD4+ T helper 1 (Th-1) cells to address the molecular bases defining cytokine
Dan Vershkov et al.
Cell reports, 26(10), 2531-2539 (2019-03-07)
Fragile X syndrome (FXS) is caused primarily by a CGG repeat expansion in the FMR1 gene that triggers its transcriptional silencing. In order to investigate the regulatory layers involved in FMR1 inactivation, we tested a collection of chromatin modulators for

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