MAB8691
Anti-Hepatitis C Virus Antibody, helicase domain NS3
Chemicon®, from mouse
Sinonimo/i:
HCV
Autenticatiper visualizzare i prezzi riservati alla tua organizzazione & contrattuali
About This Item
Codice UNSPSC:
12352203
eCl@ss:
32160702
NACRES:
NA.41
Prodotti consigliati
Origine biologica
mouse
Livello qualitativo
Forma dell’anticorpo
purified antibody
Tipo di anticorpo
primary antibodies
Clone
monoclonal
Reattività contro le specie
human
Produttore/marchio commerciale
Chemicon®
tecniche
ELISA: suitable
immunofluorescence: suitable
immunohistochemistry: suitable (paraffin)
western blot: suitable
Condizioni di spedizione
wet ice
Specificità
Specific to HCV NS3, helicase domain.
Immunogeno
Recombinant protein to NS3
Applicazioni
Anti-Hepatitis C Virus Antibody, helicase domain NS3 detects level of Hepatitis C Virus & has been published & validated for use in ELISA, IF, WB, IH(P).
ELISA
Immunohistochemistry (Parrafin embedded)
Immunofluorescence
Immunoblot
Final dilutions must be determined by end user
Immunohistochemistry (Parrafin embedded)
Immunofluorescence
Immunoblot
Final dilutions must be determined by end user
Research Category
Infectious Diseases
Infectious Diseases
Research Sub Category
Infectious Diseases - Viral
Infectious Diseases - Viral
Stato fisico
Format: Purified
Liquid in PBS, pH 7.4 containing no preservatives
Stoccaggio e stabilità
Store at -20°C in undiluted aliquots for up to 6 months from date of receipt; avoid repeated freeze-thaw cycles; short-term storage (1 week) can be at 2-8°C. Use with sterile tips to prevent contamination.
Note legali
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
Esclusione di responsabilità
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Codice della classe di stoccaggio
12 - Non Combustible Liquids
Classe di pericolosità dell'acqua (WGK)
WGK 2
Punto d’infiammabilità (°F)
Not applicable
Punto d’infiammabilità (°C)
Not applicable
Certificati d'analisi (COA)
Cerca il Certificati d'analisi (COA) digitando il numero di lotto/batch corrispondente. I numeri di lotto o di batch sono stampati sull'etichetta dei prodotti dopo la parola ‘Lotto’ o ‘Batch’.
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I documenti relativi ai prodotti acquistati recentemente sono disponibili nell’Archivio dei documenti.
Jun Zhang et al.
Molecular medicine reports, 12(4), 5109-5115 (2015-07-08)
Chronic hepatitis C virus (HCV) infection is a major cause of hepatocellular carcinoma (HCC) worldwide. The HCV non‑structural protein 3 (NS3) protein is considered to affect normal cellular functions and to be involved in HCV carcinogenesis. The expression of microRNA
Shao-Ru Chen et al.
Molecules (Basel, Switzerland), 21(10) (2016-10-19)
Hepatitis C virus (HCV) infects 200 million people worldwide, and 75% of HCV cases progress into chronic infections, which consequently cause cirrhosis and hepatocellular carcinoma. HCV infection is treated with currently considered standard drugs, including direct anti-viral agents (DAAs), alone
Ming Chen et al.
Microbiology and immunology, 60(6), 407-417 (2016-04-16)
Hepatitis C virus (HCV) non-structural protein 5A (NS5A) is a multifunctional protein that is involved in the HCV life cycle and pathogenesis. In this study, a host protein(s) interacting with NS5A by tandem affinity purification were searched for with the
Rheza Gandi Bawono et al.
The Kobe journal of medical sciences, 67(2), E38-E47 (2021-11-20)
We previously reported that hepatitis C virus (HCV) NS5A (1b, Con1) protein accepts covalent ISG15 conjugation at specific lysine (Lys) residues (K44, K68, K166, K215 and K308), exhibiting proviral effects on HCV RNA replication. Here we investigated a role of
Lin Deng et al.
Journal of virology, 96(6), e0181121-e0181121 (2022-01-20)
We previously reported that hepatitis C virus (HCV) infection activates the reactive oxygen species (ROS)/c-Jun N-terminal kinase (JNK) signaling pathway. However, the roles of ROS/JNK activation in the HCV life cycle remain unclear. We sought to identify a novel role
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