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Merck

G5146

Sigma-Aldrich

D -(+)-葡萄糖

Hybri-Max, powder, BioReagent, suitable for hybridoma

别名:

葡萄糖

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About This Item

经验公式(希尔记法):
C6H12O6
CAS号:
分子量:
180.16
Beilstein:
1724615
EC號碼:
MDL號碼:
分類程式碼代碼:
12352201
PubChem物質ID:
NACRES:
NA.75

生物源

maize

品質等級

等級

Hybri-Max

產品線

BioReagent

化驗

≥99.5%

形狀

powder

技術

cell culture | hybridoma: suitable

雜質

endotoxin, tested

mp

150-152 °C (lit.)

溶解度

H2O: 2g + 15 mL (Tested)

SMILES 字串

OC[C@H]1O[C@H](O)[C@H](O)[C@@H](O)[C@@H]1O

InChI

1S/C6H12O6/c7-1-2-3(8)4(9)5(10)6(11)12-2/h2-11H,1H2/t2-,3-,4+,5-,6+/m1/s1

InChI 密鑰

WQZGKKKJIJFFOK-DVKNGEFBSA-N

基因資訊

human ... PYGM(5837)

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一般說明

糖原磷酸化酶 (PYG) 以脑和肌肉亚型存在。在星形胶质细胞和神经元中分别表达。PYGM 基因定位于人类染色体 11q13.1

應用

D -(+)-葡萄糖已用于细胞工程。也可用于口服葡萄糖耐量测试。

生化/生理作用

糖原磷酸化酶,肌肉相关 (PYGM),是糖原分解的重要因素。精神分裂症患者 PYGM 基因表达下调。PYGM 突变导致麦卡德尔病,一种糖原贮积症。PYGM 基因与能量产生显著相关。

準備報告

如果配制浓度高于2g + 15ml,该材料可能会形成淡黄色溶液。

法律資訊

Hybri-Max is a trademark of Sigma-Aldrich Co. LLC

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 1

個人防護裝備

Eyeshields, Gloves, type N95 (US)


历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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访问文档库

Novel variant in the PYGM gene causing late-onset limb-girdle myopathy, ptosis, and camptocormia
Cheraud C, et al.
Muscle and Nerve, 57(1), 157-160 (2018)
The glial phosphorylase of glycogen isoform is reduced in the dorsolateral prefrontal cortex in chronic schizophrenia
Pinacho R, et al.
Schizophrenia Research, 177(1-3), 37-43 (2016)
CDKN1C/p57 kip2 is a candidate tumor suppressor gene in human breast cancer
Larson PS, et al.
BMC Cancer, 8(1), 68-68 (2008)
Comparative metabolic analysis of CHO cell clones obtained through cell engineering, for IgG productivity, growth and cell longevity
Wilkens CA and Gerdtzen ZP
PLoS ONE, 10(3) (2015)
Electrical vs manual acupuncture stimulation in a rat model of polycystic ovary syndrome: different effects on muscle and fat tissue insulin signaling
Johansson J, et al.
PLoS ONE, 8(1) (2013)

商品

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We presents an article about the Warburg effect, and how it is the enhanced conversion of glucose to lactate observed in tumor cells, even in the presence of normal levels of oxygen. Otto Heinrich Warburg demonstrated in 1924 that cancer cells show an increased dependence on glycolysis to meet their energy needs, regardless of whether they were well-oxygenated or not.

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