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Merck

EM0200

Sigma-Aldrich

Osteo-Bed骨包埋试剂盒

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About This Item

分類程式碼代碼:
12171500
NACRES:
NA.47

應用

hematology
histology

品質等級

儲存溫度

room temp

應用

Suitable for use with large and small mineralized (undecalcified) bone sections. Yields clear, hard blocks for cutting sections. Not water-soluble.

推薦產品

Osteo-Bed Bone Embedding Solvent (O8639) is available to remove plastic from sections.

仅试剂盒组分

产品编号
说明

  • Osteo-Bed Bone Embedding Resin A (O8514) 900 mL

  • Osteo-Bed Bone Embedding Catalyst (O8764) 2 x 12

推薦

产品编号
说明
价格

訊號詞

Danger

危險分類

Aquatic Acute 1 - Aquatic Chronic 1 - Eye Irrit. 2 - Flam. Liq. 2 - Org. Perox. D - Repr. 1B - Skin Irrit. 2 - Skin Sens. 1 - STOT SE 3

標靶器官

Respiratory system

儲存類別代碼

5.2 - Organic peroxides and self-reacting hazardous materials

閃點(°F)

50.0 °F

閃點(°C)

10 °C


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Colin K Yee et al.
Clinical & experimental metastasis, 25(8), 903-911 (2008-09-25)
In the current study, we examine heparin's anti-metastatic properties by using a well-defined mouse model of osteolytic bone metastasis. C57BL/6 mice were treated with increasing doses of unfractionated heparin (15, 20, or 25 units/mouse) 30 min prior to the left
Nur-Vaizura Mohamad et al.
International journal of medical sciences, 18(16), 3665-3673 (2021-11-19)
Menopause is the leading cause of osteoporosis for elderly women due to imbalanced bone remodelling in the absence of oestrogen. The ability of tocotrienol in reversing established bone loss due to oestrogen deficiency remains unclear despite the plenitude of evidence
Xuekun Fu et al.
Signal transduction and targeted therapy, 5(1), 297-297 (2020-12-29)
In vertebrates, the type 1 parathyroid hormone receptor (PTH1R) is a critical regulator of skeletal development and homeostasis; however, how it is modulated is incompletely understood. Here we report that deleting Kindlin-2 in osteoblastic cells using the mouse 10-kb Dmp1-Cre
Yishu Wang et al.
JCI insight, 4(22) (2019-11-15)
Mammalian focal adhesion proteins Pinch1 and Pinch2 regulate integrin activation and cell-extracellular matrix adhesion and migration. Here, we show that deleting Pinch1 in osteocytes and mature osteoblasts using the 10-kb mouse Dmp1-Cre and Pinch2 globally (double KO; dKO) results in

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