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Merck

T27006

Sigma-Aldrich

2-噻吩甲酰三氟丙酮

99%

别名:

4,4,4-三氟-1-(2-噻吩基)-1,3-丁二酮, TTA(萃取剂), TTB, TTFA

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About This Item

经验公式(希尔记法):
C8H5F3O2S
CAS号:
分子量:
222.18
Beilstein:
168645
EC號碼:
MDL號碼:
分類程式碼代碼:
12352100
PubChem物質ID:
NACRES:
NA.22

化驗

99%

bp

96-98 °C/8 mmHg (lit.)

mp

40-44 °C (lit.)

儲存溫度

2-8°C

SMILES 字串

FC(F)(F)C(=O)CC(=O)c1cccs1

InChI

1S/C8H5F3O2S/c9-8(10,11)7(13)4-5(12)6-2-1-3-14-6/h1-3H,4H2

InChI 密鑰

TXBBUSUXYMIVOS-UHFFFAOYSA-N

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應用

用于锕系元素和镧系元素的测定。

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 3

閃點(°F)

233.6 °F - closed cup

閃點(°C)

112 °C - closed cup

個人防護裝備

dust mask type N95 (US), Eyeshields, Gloves


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Jeffery, G.H. et al.
Vogel's Textbook of Quantitative Chemical Analysis, 170-170 (1989)
Vladimir A Kokorekin et al.
Molecules (Basel, Switzerland), 25(18) (2020-09-17)
In this article, we demonstrate how an original effective "metal-free" and "chromatography-free" route for the synthesis of 3-thiocyanatopyrazolo[1,5-a]pyrimidines has been developed. It is based on electrooxidative (anodic) C-H thiocyanation of 5-aminopyrazoles by thiocyanate ion leading to 4-thiocyanato-5-aminopyrazoles (stage 1, yields
Yajuan Li et al.
Dalton transactions (Cambridge, England : 2003), 39(10), 2554-2562 (2010-02-25)
A series of novel ternary organic-inorganic mesoporous polymeric hybrids TTFA-S16-Eu-PMMA, TTFA-S16-Eu-PMAA, and TTFA-S16-Eu-PVP (TTFA = 2-Thenoyltrifluoroacetone; PMMA = polymethyl methacrylate; PMAA = polymethacrylic acid; PVP = polyvinylpyrrolidone) have been assembled by the Eu(3+) complex covalently attaching to the TTFA directly
Yongqiang Chen et al.
Journal of cell science, 120(Pt 23), 4155-4166 (2007-11-23)
Autophagy is a self-digestion process important for cell survival during starvation. It has also been described as a form of programmed cell death. Mitochondria are important regulators of autophagy-induced cell death and damaged mitochondria are often degraded by autophagosomes. Inhibition
Stefan Dröse et al.
Molecular pharmacology, 79(5), 814-822 (2011-02-01)
In this study, we have analyzed the effect of different cardioprotective complex II inhibitors on the mitochondrial production of reactive oxygen species (ROS) because ROS seem to be essential for signaling during preconditioning to prevent ischemia/reperfusion injury. Despite different binding

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