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Merck
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Principaux documents

AB9272

Sigma-Aldrich

Anti-Synaptophysin Antibody

Chemicon®, from rabbit

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About This Item

Code UNSPSC :
12352203
eCl@ss :
32160702
Nomenclature NACRES :
NA.41

Source biologique

rabbit

Niveau de qualité

Forme d'anticorps

affinity purified immunoglobulin

Type de produit anticorps

primary antibodies

Clone

polyclonal

Produit purifié par

affinity chromatography

Espèces réactives

human, rat

Fabricant/nom de marque

Chemicon®

Technique(s)

immunohistochemistry: suitable (paraffin)
western blot: suitable

Numéro d'accès NCBI

Numéro d'accès UniProt

Conditions d'expédition

wet ice

Modification post-traductionnelle de la cible

unmodified

Informations sur le gène

human ... SYP(6855)

Spécificité

Synaptophysin

Immunogène

Synthetic peptide from human synaptophysin.

Application

Anti-Synaptophysin Antibody detects level of Synaptophysin & has been published & validated for use in WB, IH(P).
Research Category
Neuroscience
Research Sub Category
Synapse & Synaptic Biology
Western blot

Immunohistochemistry on formalin fixed, paraffin embedded tissue: 1:300 for 10 minutes at room temperature. Staining of formalin-fixed tissues requires boiling of tissues in 10 mM citrate buffer, pH 6.0 for 10 minutes followed by cooling at room temperature for 20 minutes.

Optimal working dilutions must be determined by the end user.

STAINING PATTERN: Cytoplasmic

Liaison

Replaces: 04-1019

Forme physique

Affinity purified immunoglobulin. Liquid in 10 mM PBS, pH 7.4 containing BSA and 0.1% sodium azide.

Stockage et stabilité

Maintain at 2-8°C for up to 6 months after date of receipt.

Remarque sur l'analyse

Control
POSITIVE CONTROL: Pancreas or Pheochromocytoma

Informations légales

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Clause de non-responsabilité

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Code de la classe de stockage

12 - Non Combustible Liquids

Classe de danger pour l'eau (WGK)

WGK 2

Point d'éclair (°F)

Not applicable

Point d'éclair (°C)

Not applicable


Certificats d'analyse (COA)

Recherchez un Certificats d'analyse (COA) en saisissant le numéro de lot du produit. Les numéros de lot figurent sur l'étiquette du produit après les mots "Lot" ou "Batch".

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Consulter la Bibliothèque de documents

Rachel Comba et al.
F1000Research, 4, 396-396 (2016-03-01)
Improvement on spatial tasks is observed during a late, postnatal developmental period (PND18 - PND24).  The purpose of the current work was 1) to determine whether the emergence of spatial-behavioral function was based on the ability to generate appropriate behavioral
Blake R McAlpin et al.
Theranostics, 12(2), 603-619 (2022-01-04)
Breast cancer is the most common female malignancy in both the developed and developing world. Doxorubicin is one of the most commonly used chemotherapies for breast cancer. Unfortunately, up to 60% of survivors report long-term chemotherapy-induced cognitive dysfunction (CICD) characterized
Luise Appeltshauser et al.
Neurology(R) neuroimmunology & neuroinflammation, 9(3) (2022-03-23)
Nodo-paranodopathies are peripheral neuropathies with dysfunction of the node of Ranvier. Affected patients who are seropositive for antibodies against adhesion molecules like contactin-1 and neurofascin show distinct clinical features and a disruption of the paranodal complex. An axoglial dysjunction is
GABA(B) receptor activation triggers BDNF release and promotes the maturation of GABAergic synapses.
Fiorentino, H; Kuczewski, N; Diabira, D; Ferrand, N; Pangalos, MN; Porcher, C; Gaiarsa, JL
The Journal of Neuroscience null
Goran Tomic et al.
Cell stem cell, 23(3), 436-443 (2018-08-14)
The intestinal epithelium is largely maintained by self-renewing stem cells but with apparently committed progenitors also contributing, particularly following tissue damage. However, the mechanism of, and requirement for, progenitor plasticity in mediating pathological response remain unknown. Here we show that phosphorylation

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