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SAB5600204

Sigma-Aldrich

Anti-β-Actin (ACTB) Antibody

rabbit monoclonal, RM112

Sinonimo/i:

ACTB, Actin cytoplastic 1, actin beta, beta-actin

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About This Item

Codice UNSPSC:
12352203
NACRES:
NA.41

Nome del prodotto

Anti-beta-Actin antibody, Rabbit monoclonal, recombinant, expressed in HEK 293 cells, clone RM112, purified immunoglobulin

Ricombinante

expressed in HEK 293 cells

Forma dell’anticorpo

purified immunoglobulin

Tipo di anticorpo

primary antibodies

Clone

RM112, monoclonal
recombinant monoclonal

Stato

buffered aqueous glycerol solution

Reattività contro le specie

all

tecniche

ChIP: 1:200
ELISA: suitable (assay dependent concentration)
flow cytometry: suitable (assay dependent concentration)
immunocytochemistry: 1:200
immunohistochemistry: 1:200
immunoprecipitation (IP): 1:200
western blot: 1:1000

Isotipo

IgG

N° accesso NCBI

N° accesso UniProt

Condizioni di spedizione

wet ice

Temperatura di conservazione

−20°C

modifica post-traduzionali bersaglio

unmodified

Informazioni sul gene

human ... ACTB(60)

Specificità

This antibody reacts to beta-Actin (Actin, cytoplasmic 1). No cross reactivity with other isoforms of Actin.

Immunogeno

A peptide corresponding to the N-terminus of beta-actin

Stato fisico

Solution in phosphate buffered saline containing 50% Glycerol/PBS with 1% BSA and 0.09% sodium azide.

Esclusione di responsabilità

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Codice della classe di stoccaggio

10 - Combustible liquids

Classe di pericolosità dell'acqua (WGK)

WGK 2

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable


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During retinal development, a physiologic hypoxia stimulates endothelial cell proliferation. The hypoxic milieu warrants retina vascularization and promotes the activation of several mechanisms aimed to ensure homeostasis and energy balance of both endothelial and retinal cells. Autophagy is an evolutionarily
Joselyn Padilla et al.
Cells, 11(7) (2022-04-13)
The oncogenic expression or mutation of tumor suppressors drives metabolic alteration, causing cancer cells to utilize diverse nutrients. Lactate is a known substrate for cancer cells, yet the regulatory mechanisms of lactate catabolism are limited. Here, we show that a

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