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CdO/CdA

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GF10529603

Iron

Name: Iron
Brand: ALDRICH

rod, 50mm, diameter 10.0mm, as drawn, 99.99+%

Sinonimo/i:

Iron, FE007955

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About This Item

Formula condensata:

Numero CAS:

7439-89-6

Peso molecolare:

55.85

Numero MDL:

MFCD00010999

Codice UNSPSC:

12352300

ID PubChem:

329801656

NACRES:

NA.23

Saggio

99.99%

Forma fisica

rod

Produttore/marchio commerciale

Goodfellow 105-296-03

Resistività

9.71 μΩ-cm

L × diam.

50 mm × 10.0 mm

P. eboll.

2750 °C (lit.)

Punto di fusione

1535 °C (lit.)

Densità

7.86 g/mL at 25 °C (lit.)

Stringa SMILE

[Fe]

InChI

1S/Fe
XEEYBQQBJWHFJM-UHFFFAOYSA-N

Categorie correlate

Metalli e leghe ad elevata purezza

Descrizione generale

For updated SDS information please visit www.goodfellow.com.

Note legali

Product of Goodfellow

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Aerobactin mediates virulence and accounts for increased siderophore production under iron-limiting conditions by hypervirulent (hypermucoviscous) Klebsiella pneumoniae.

Thomas A Russo et al.

Infection and immunity, 82(6), 2356-2367 (2014-03-26)

Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our

Crystal structure of PhnZ in complex with substrate reveals a di-iron oxygenase mechanism for catabolism of organophosphonates.

Laura M van Staalduinen et al.

Proceedings of the National Academy of Sciences of the United States of America, 111(14), 5171-5176 (2014-04-08)

The enzymes PhnY and PhnZ comprise an oxidative catabolic pathway that enables marine bacteria to use 2-aminoethylphosphonic acid as a source of inorganic phosphate. PhnZ is notable for catalyzing the oxidative cleavage of a carbon-phosphorus bond using Fe(II) and dioxygen

The iron-sensing fur regulator controls expression timing and levels of salmonella pathogenicity island 2 genes in the course of environmental acidification.

Eunna Choi et al.

Infection and immunity, 82(6), 2203-2210 (2014-03-20)

In order to survive inside macrophages, Salmonella produces a series of proteins encoded by genes within Salmonella pathogenicity island 2 (SPI-2). In the present study, we report that Fur, a central regulator of iron utilization, negatively controls the expression of

Hypoferremia of infection: a double-edged sword?

Kristen L Lokken et al.

Nature medicine, 20(4), 335-337 (2014-04-09)

Inverse agonist of estrogen-related receptor γ controls Salmonella typhimurium infection by modulating host iron homeostasis.

Don-Kyu Kim et al.

Nature medicine, 20(4), 419-424 (2014-03-25)

In response to microbial infection, expression of the defensin-like peptide hepcidin (encoded by Hamp) is induced in hepatocytes to decrease iron release from macrophages. To elucidate the mechanism by which Salmonella enterica var. Typhimurium (S. typhimurium), an intramacrophage bacterium, alters

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