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Key Documents

AB9482

Sigma-Aldrich

Anti-Retinoid-Related Orphan Receptor β Antibody

Chemicon®, from rabbit

Synonym(e):

ROR Beta/NR

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About This Item

UNSPSC-Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

Biologische Quelle

rabbit

Qualitätsniveau

Antikörperform

affinity purified immunoglobulin

Antikörper-Produkttyp

primary antibodies

Klon

polyclonal

Aufgereinigt durch

affinity chromatography

Speziesreaktivität

human

Hersteller/Markenname

Chemicon®

Methode(n)

immunohistochemistry: suitable (paraffin)

NCBI-Hinterlegungsnummer

UniProt-Hinterlegungsnummer

Versandbedingung

dry ice

Posttranslationale Modifikation Target

unmodified

Angaben zum Gen

human ... RORB(6096)

Spezifität

Recognizes Retinoid-Related Orphan Receptor Beta [ROR Beta/NR].

Immunogen

Synthetic peptide from the hinge domain of human Retinoid-Related Orphan Receptor Beta.

Anwendung

Research Category
Epigenetik & nukleäre Funktionen
Research Sub Category
Transkriptionsfaktoren
Immunohistochemistry on paraffin embedded tissue sections.

Optimal working dilutions must be determined by the end user.
Use Anti-Retinoid-Related Orphan Receptor β Antibody (Rabbit Polyclonal Antibody) validated in IHC(P) to detect Retinoid-Related Orphan Receptor β also known as ROR Beta/NR.

Physikalische Form

Affinity purified immunoglobulin. Liquid in PBS, pH 7.7 with 0.01% sodium azide.

Lagerung und Haltbarkeit

Maintain at -70°C in undiluted aliquots for up to 6 months after date of receipt. Avoid repeated freeze/thaw cycles.

Sonstige Hinweise

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

Rechtliche Hinweise

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Haftungsausschluss

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Lagerklassenschlüssel

10 - Combustible liquids

WGK

WGK 2

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable


Analysenzertifikate (COA)

Suchen Sie nach Analysenzertifikate (COA), indem Sie die Lot-/Chargennummer des Produkts eingeben. Lot- und Chargennummern sind auf dem Produktetikett hinter den Wörtern ‘Lot’ oder ‘Batch’ (Lot oder Charge) zu finden.

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Die Dokumentenbibliothek aufrufen

Claudia Günther et al.
The Journal of clinical investigation, 126(11), 4346-4360 (2016-11-02)
Although necrosis and necroinflammation are central features of many liver diseases, the role of programmed necrosis in the context of inflammation-dependent hepatocellular death remains to be fully determined. Here, we have demonstrated that the pseudokinase mixed lineage kinase domain-like protein
The pathogenic role of the canonical Wnt pathway in age-related macular degeneration.
Zhou, T; Hu, Y; Chen, Y; Zhou, KK; Zhang, B; Gao, G; Ma, JX
Investigative Ophthalmology & Visual Science null
Contrasting behavior of the p18INK4c and p16INK4a tumor suppressors in both replicative and oncogene-induced senescence.
Gagrica, S; Brookes, S; Anderton, E; Rowe, J; Peters, G
Cancer Research null
Vincent Ricchiuti et al.
The Journal of endocrinology, 211(1), 47-54 (2011-07-13)
Liberal or high-sodium (HS) intake, in conjunction with an activated renin-angiotensin-aldosterone system, increases cardiovascular (CV) damage. We tested the hypothesis that sodium intake regulates the type 1 angiotensin II receptor (AT(1)R), mineralocorticoid receptor (MR), and associated signaling pathways in heart
Yaling Yin et al.
Proceedings of the National Academy of Sciences of the United States of America, 113(26), E3773-E3781 (2016-06-15)
Intracellular accumulation of wild-type tau is a hallmark of sporadic Alzheimer's disease (AD), but the molecular mechanisms underlying tau-induced synapse impairment and memory deficit are poorly understood. Here we found that overexpression of human wild-type full-length tau (termed hTau) induced

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