84885
pI-Select
set of 10 acrylamido buffer solutions (10 ml each) pK 1.0-12
Sinónimos:
Acrylamido buffers, set
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About This Item
Productos recomendados
Quality Level
form
liquid
shelf life
limited shelf life
pKa
1.0
10.3
≥12.0
3.1
3.6
4.6
6.2
7.0
8.5
9.3
storage temp.
2-8°C
Other Notes
Focusing of alkaline proteases (subtilisins) in pH 10-12 immobilized gradients
signalword
Danger
hcodes
Hazard Classifications
Eye Dam. 1 - Flam. Liq. 2 - STOT SE 3
target_organs
Central nervous system
Storage Class
3 - Flammable liquids
flash_point_f
71.6 °F - closed cup
flash_point_c
22 °C - closed cup
ppe
Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter
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Retinoblastoma (RB) represents an aggressive malignancy in the eye during the period of infancy and childhood. We delineated the ability of microRNA-186 (miR-186) to influence viability, invasion, migration, angiogenesis, and apoptosis of RB via the Hedgehog signaling pathway by targeting
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Myocardial ischemia-reperfusion injury (MIRI) is recognized as a major cause of morbidity and mortality which is commonly associated with coronary artery disease. In recent studies, annexin A1 gene (ANXA1) has been discovered to be involved in the treatment for MIRI.
Electrophoresis, 15(12), 1535-1540 (1994-12-01)
Isoelectric focusing in very alkaline immobilized pH gradients (IPG) was adopted for checking the purity and assessing the pI value of two strongly alkaline proteases: Savinase and Durazym. The first enzyme (known to be the most alkaline) contains 5 Asp
American journal of physiology. Cell physiology, 317(2), C189-C199 (2019-05-03)
Sepsis is a systemic inflammatory response syndrome resulting from infection. This study aimed at exploring the role of microRNA-140 (miR-140) in septic mice. Wnt family member 11 (WNT11) was verified to be a target gene of miR-140 after bioinformatic prediction
Frontiers in physiology, 11, 551318-551318 (2020-11-17)
Myocardial ischemia-reperfusion (MI/R) injury is characterized by iron deposition and reactive oxygen species production, which can induce ferroptosis. Ferroptosis has also been proposed to promote cardiomyocyte death. The current study sought to define the mechanism governing cardiomyocyte death in MI/R
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