810528P
Avanti
16:0 DNP Cap PE
1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine-N-[6-[(2,4-dinitrophenyl)amino]hexanoyl] (ammonium salt), powder
Sinónimos:
1,2-dihexadecanoyl-sn-glycero-3-phosphoethanolamine-N-[6-[(2,4-dinitrophenyl)amino]caproyl] (ammonium salt)
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About This Item
Productos recomendados
assay
>99% (TLC)
form
powder
packaging
pkg of 1 × 25 mg (810528P-25mg)
manufacturer/tradename
Avanti Research™ - A Croda Brand 810528P
shipped in
dry ice
storage temp.
−20°C
Categorías relacionadas
General description
Phosphoethanolamine is a precursor to phosphatidylcholine and phosphatidylethanolamine. 2,4-dinitrophenol (DNP) is a synthetic lipid-soluble compound which transports ions across the bioenergetic membranes.
Application
16:0 DNP Cap PE may be used in Rho-PE-doped ink solution for the cell recruiting based lipid array experiments.
Biochem/physiol Actions
Phosphoethanolamine participates in phospholipid metabolism. It has the ability to repress tumor growth both in vitro and in vivo.
Packaging
5 mL Clear Glass Sealed Ampule (810528P-25mg)
Legal Information
Avanti Research is a trademark of Avanti Polar Lipids, LLC
Storage Class
11 - Combustible Solids
Certificados de análisis (COA)
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Membrane Transport
An Introduction to Biological Membranes, 423-451 (2016)
ACS nano (2019-12-31)
Activation of membrane receptors through clustering is a common mechanism found in various biological systems. Spatial proximity of receptors may be transduced across the membrane to initiate signaling pathways or alternatively be recognized by peripheral proteins or immune cells to
Ecancermedicalscience, 10, 681-681 (2016-12-03)
The use of unproven forms of therapy in cancer treatment is very common. In Brazil, the distribution by researchers to patients of an investigational agent called phophoethanolamine (PHOS) has led to a widely publicized scientific scandal. PHOS is a precursor
Clinical and experimental immunology (2019-12-20)
Carbamylation is a post-translational modification that can be detected on a range of proteins, including immunoglobulin (Ig)G, in several clinical conditions. Carbamylated IgG (ca-IgG) was reported to lose its capacity to trigger complement activation, but the mechanism remains unclear. Because
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