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Merck
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重要文件

RAB1177

Sigma-Aldrich

小鼠TSP-2 ELISA试剂盒

for cell culture supernatants, plasma, and serum samples

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About This Item

分類程式碼代碼:
41116158
NACRES:
NA.32

物種活性

mouse

包裝

kit of 96 wells (12 strips x 8 wells)

技術

ELISA: suitable

輸入

sample type serum sample(s)
sample type plasma
sample type cell culture supernatant(s)

assay range

inter-assay cv: <12%
intra-assay cv: <10%
sensitivity: 0.16 ng/ml

檢測方法

colorimetric

運輸包裝

wet ice

儲存溫度

−20°C

基因資訊

mouse ... Thbs2(21826)

一般說明

This ELISA antibody pair detects Mouse Thrombospondin 2 (THBS2)

應用

For research use only. Not for use in diagnostic procedures.
Please refer to the attached Protocolfor details.

生化/生理作用

Thrombospondin-2 (TSP2) is an extracellular matrix (ECM) protein involved in regulating fracture healing. In addition, it also regulates cell-matrix interactions. TSP2 plays a vital role in assembly of collagen fibrils in the skin. It is involved in tissue repair and foreign body responses. The matricellular protein TSP2 endogenously controls angiogenesis and inflammation in rheumatoid arthritis patients. It also acts as an endogenous inhibitor of tumor growth.

其他說明

A sample Certificate of Analysis is available for this product. Please type the word sample in the text box provided for lot number.

象形圖

Corrosion

訊號詞

Warning

危險聲明

防範說明

危險分類

Met. Corr. 1

儲存類別代碼

8A - Combustible corrosive hazardous materials

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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分析證明 (COA)

Lot/Batch Number

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存取文件庫

Thrombospondin-2: a potent endogenous inhibitor of tumor growth and angiogenesis.
Streit M, et al.
Proceedings of the National Academy of Sciences of the USA, 96(26), 14888-14893 (1999)
Thrombospondin?2 influences the proportion of cartilage and bone during fracture healing.
Taylor D K, et al.
Journal of Bone and Mineral Research, 24(6), 1043-1054 (2009)
The functions of thrombospondin-1 and-2.
Lawler J
Current Opinion in Cell Biology, 12(5), 634-640 (2000)
Thrombospondin 2 functions as an endogenous regulator of angiogenesis and inflammation in rheumatoid arthritis.
Park Y W, et al.
The American Journal of Pathology, 165(6), 2087-2098 (2004)

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