推薦產品
一般說明
人副流感病毒3型（PI3病毒）可引起婴儿轻度至重度呼吸道感染。PI3病毒是一种包膜RNA病毒，其外表面具有两种糖蛋白：血凝素-神经氨酸酶（HN）和融合（F）。已知这两种糖蛋白负责感染过程的开始和进展
免疫原
人副流感病毒3型融合糖蛋白
法律資訊
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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儲存類別代碼
10 - Combustible liquids
水污染物質分類（WGK）
WGK 1
閃點（°F）
Not applicable
閃點（°C）
Not applicable
分析證明 (COA)
輸入產品批次/批號來搜索 分析證明 (COA)。在產品’s標籤上找到批次和批號，寫有 ‘Lot’或‘Batch’.。
M A Horga et al.
Journal of virology, 74(24), 11792-11799 (2000-11-23)
Viral interference is characterized by the resistance of infected cells to infection by a challenge virus. Mechanisms of viral interference have not been characterized for human parainfluenza virus type 3 (HPF3), and the possible role of the neuraminidase (receptor-destroying) enzyme
Antiviral effects of glycosylation and glucose trimming inhibitors on human parainfluenza virus type 3
Tanaka, Yoshikazu, et al
Antiviral research, 72, 1-9 (2006)
Laura M Palermo et al.
Journal of virology, 83(13), 6900-6908 (2009-04-24)
Three discrete activities of the paramyxovirus hemagglutinin-neuraminidase (HN) protein, receptor binding, receptor cleaving (neuraminidase), and triggering of the fusion protein, each affect the promotion of viral fusion and entry. For human parainfluenza virus type 3 (HPIV3), the effects of specific
Wenyan Xie et al.
PloS one, 10(8), e0136474-e0136474 (2015-08-26)
Human parainfluenza virus type 3 (HPIV3) can cause severe respiratory tract diseases in infants and young children, but no licensed vaccines or antiviral agents are currently available for treatment. Fusing the viral and target cell membranes is a prerequisite for
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