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Key Documents

07-748

Sigma-Aldrich

Anti-monomethyl-Histone H4 (Lys20) Antibody

Upstate®, from rabbit

同義詞:

H4K20me1, Histone H4 (mono methyl K20)

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About This Item

分類程式碼代碼:
12352203
eCl@ss:
32160702
NACRES:
NA.41

生物源

rabbit

品質等級

抗體表格

purified antibody

抗體產品種類

primary antibodies

無性繁殖

polyclonal

物種活性

human

製造商/商標名

Upstate®

技術

immunocytochemistry: suitable
multiplexing: suitable
western blot: suitable

同型

IgG

NCBI登錄號

UniProt登錄號

運輸包裝

wet ice

目標翻譯後修改

monomethylation (Lys20)

基因資訊

特異性

Broad species cross-reactivity expected due to sequence conservation
monomethylated Histone H4 (Lys20)

免疫原

KLH-conjugated, branched synthetic peptide corresponding to amino acids 15-25 (AKRHR[me1K]VLRDN) of human Histone H4

應用

Additional Referenced Research Applications:
Immunocytochemistry (cells)
Anti-monomethyl-Histone H4 (Lys20) Antibody is a rabbit polyclonal antibody for detection of monomethyl-Histone H4 (Lys20) also known as H4K20me1, Histone H4 (mono methyl K20) & has been validated in WB, Mplex, ICC.
Research Category
Epigenetics & Nuclear Function
Research Sub Category
Histones

品質

Routinely evaluated by immunoblot

標靶描述

10kDa

外觀

Protein A purified
0.1M Tris-glycine, pH 7.4, 0.15M NaCl, 0.05% sodium azide and 30% glycerol.
Format: Purified

儲存和穩定性

2 years at -20°C from date of shipment

法律資訊

UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany

免責聲明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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儲存類別代碼

10 - Combustible liquids

水污染物質分類(WGK)

WGK 1


分析證明 (COA)

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Li Liu et al.
PloS one, 8(11), e78253-e78253 (2013-11-14)
The upstream Gγ-globin cAMP-response element (G-CRE) plays an important role in regulating Gγ-globin expression through binding of ATF2 and its DNA-binding partners defined in this study. ATF2 knockdown resulted in a significant reduction of γ-globin expression accompanied by decreased ATF2

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