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Merck

V900884

Sigma-Aldrich

十二烷基三甲基氯化铵

Vetec, reagent grade, ≥98%

别名:

N-Dodecyl-N,N,N-trimethylammonium chloride, DTAC, Trimethyldodecylammonium chloride

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About This Item

线性分子式:
CH3(CH2)11N(CH3)3Cl
CAS号:
分子量:
263.89
Beilstein:
3915951
EC號碼:
MDL號碼:
分類程式碼代碼:
12352107
PubChem物質ID:
化驗:
≥98%

等級

reagent grade

產品線

Vetec

化驗

≥98%

mp

37 °C

官能基

amine

SMILES 字串

[Cl-].CCCCCCCCCCCC[N+](C)(C)C

InChI

1S/C15H34N.ClH/c1-5-6-7-8-9-10-11-12-13-14-15-16(2,3)4;/h5-15H2,1-4H3;1H/q+1;/p-1

InChI 密鑰

DDXLVDQZPFLQMZ-UHFFFAOYSA-M

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一般說明

十二烷基三甲基氯化铵(DTAC)是一种季铵化合物(QAC),常用作阳离子表面活性剂。主要用于蛋白质变性。

應用

DTAC可用作下列反应中的阳离子表面活性剂:
  • 酮/醛的转移氢化反应。
  • 烯烃的开环复分解聚合反应。

法律資訊

Vetec is a trademark of Merck KGaA, Darmstadt, Germany

象形圖

Exclamation markEnvironment

訊號詞

Warning

危險分類

Acute Tox. 4 Oral - Aquatic Acute 1 - Eye Irrit. 2 - Skin Irrit. 2

儲存類別代碼

13 - Non Combustible Solids

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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分析证书(COA)

Lot/Batch Number

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Johannes Weirather et al.
Circulation research, 115(1), 55-67 (2014-05-03)
An exaggerated or persistent inflammatory activation after myocardial infarction (MI) leads to maladaptive healing and subsequent remodeling of the left ventricle. Foxp3(+) CD4(+) regulatory T cells (Treg cells) contribute to inflammation resolution. Therefore, Treg cells might influence cardiac healing post-MI.
Jing Shang et al.
Scientific reports, 5, 13651-13651 (2015-09-04)
In-cell immunoassays have become a valuable tool for protein expression analysis complementary to established assay formats. However, comprehensive molecular characterization of individual specimens has proven challenging and impractical due to, in part, a singleplex nature of reporter enzymes and technical
Sara G Hovakeemian et al.
Soft matter, 11(34), 6840-6851 (2015-08-04)
Most antimicrobial peptides act upon target microorganisms by permeabilizing their membranes. The mode of action is often assessed by vesicle leakage experiments that use model membranes, with the assumption that biological activity correlates with the permeabilization of the lipid bilayer.

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