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Merck
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Key Documents

T3392

Sigma-Aldrich

Testosterone 3-(O-carboxymethyl)oxime: BSA

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About This Item

CAS号:
MDL號碼:
分類程式碼代碼:
12352200
NACRES:
NA.77

化驗

≥98% (TLC)

品質等級

形狀

lyophilized powder

藥物控制

regulated under CDSA - not available from Sigma-Aldrich Canada

標籤範圍

20-30 mol steroid per mol BSA

溶解度

phosphate buffer: 1.90-2.10 mg/mL, clear to slightly hazy, colorless (pH 7.50)

運輸包裝

ambient

儲存溫度

2-8°C

InChI

1S/C21H31NO4/c1-20-9-7-14(22-26-12-19(24)25)11-13(20)3-4-15-16-5-6-18(23)21(16,2)10-8-17(15)20/h11,15-18,23H,3-10,12H2,1-2H3,(H,24,25)/t15-,16-,17-,18-,20-,21-/m0/s1

InChI 密鑰

VDYLVWGBLQNNAW-ZKHIMWLXSA-N

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

Eyeshields, Gloves, type N95 (US)


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Yizhou Zhang et al.
Aging, 11(8), 2281-2294 (2019-04-22)
The non-genomic actions of androgen-induced synaptic plasticity have been extensively studied. However, the underlying mechanisms remain controversial. We recently found that testosterone-fetal bovine serum albumin (T-BSA), a cell membrane-impermeable complex, led to a rapid increase in the postsynaptic density 95
Sha Li et al.
Molecular and cellular endocrinology, 414, 82-90 (2015-07-15)
Testosterone (T), the principal androgen, and its metabolite, dihydrotestosterone (DHT), are known to mediate their effects through binding to intracellular androgen receptors (iARs). In addition to their well-known genomic effects, androgens rapidly alter neuronal excitability through a non-genomic pathway mediated

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