等級
for molecular biology
品質等級
描述
non-ionic
(application(s): life science and biopharma)
種類
Type 15-S-9
形狀
liquid
分子量
596 g/mol
環保替代產品特色
Design for Degradation
Learn more about the Principles of Green Chemistry.
sustainability
Greener Alternative Product
CMC
52 ppm
轉變溫度
cloud point 60 °C
pour point 9 °C
HLB
13.3
異物活動
foreign activity (Dnase, Rnase, none detected)
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相关类别
一般說明
We are committed to bringing you Greener Alternative Products, which adhere to one or more of The 12 Principles of Green Chemistry. This product is a biodegradable surfactant and is aligned with the 10th principle of Green Chemistry "Design for Degradation".
Tergitol™ 15-S-9 is a linear non-ionic surfactant that is a secondary ethoxylated alcohol. This clear liquid surfactant is compatible with anionic, cationic, and other nonionic surfactants.
Tergitol™ 15-S-9 is a linear non-ionic surfactant that is a secondary ethoxylated alcohol. This clear liquid surfactant is compatible with anionic, cationic, and other nonionic surfactants.
應用
Tergitol has been used as a surfactant to study its influence on the formation of multi-phase micro-emulsions.
Tergitol™ 15-S-9 has been used:
- To synthesize thermally stable mesoporous alumina
- To synthesize porous TiO2
- Along with SDS, ethanol and water for continuous phase preparations in electrospray emulsification
- In the synthesis of Lath-like γ-alumina and boehmite nanofibers
特點和優勢
- Superior detergency
- Rapid dissolution & good rinseability
- Excellent formulation & handling properties
- Narrow gel range
- Readily biodegradable (OECD 301E)
- Low odor
其他說明
聚乙二醇醚(非离子型)表面活性剂。
法律資訊
TERGITOL is a trademark of The Dow Chemical Company ("Dow") or an affiliated company of Dow
訊號詞
Danger
危險分類
Acute Tox. 4 Inhalation - Acute Tox. 4 Oral - Eye Dam. 1 - Skin Irrit. 2
儲存類別代碼
10 - Combustible liquids
水污染物質分類(WGK)
WGK 3
Interfacial tension and phase behavior of surfactant-brine?oil system.
Colloids and Surfaces. A, Physicochemical and Engineering Aspects, 383(1-3), 114-119 (2011)
Nature metabolism, 2(12), 1373-1381 (2020-11-25)
The oncogenic KRAS mutation has a critical role in the initiation of human pancreatic ductal adenocarcinoma (PDAC) since it rewires glutamine metabolism to increase reduced nicotinamide adenine dinucleotide phosphate (NADPH) production, balancing cellular redox homeostasis with macromolecular synthesis1,2. Mitochondrial glutamine-derived
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