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Merck
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Key Documents

DR1001

Sigma-Aldrich

Anti-8-Hydroxyguanosine Goat pAb

liquid, Calbiochem®

别名:

Anti-8-OHG, Anti-8-oxo-G

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About This Item

分類程式碼代碼:
12352203
NACRES:
NA.43

生物源

goat

品質等級

100

抗體表格

serum

抗體產品種類

primary antibodies

無性繁殖

polyclonal

形狀

liquid

不包含

preservative

物種活性（以同源性預測）

all

製造商／商標名

Calbiochem®

儲存條件

OK to freeze
avoid repeated freeze/thaw cycles

同型

IgG

運輸包裝

wet ice

儲存溫度

−20°C

目標翻譯後修改

unmodified

一般說明

Goat polyclonal antibody supplied as undiluted serum. Recognizes 8-hydroxydeoxy-guanosine.
Recognizes 8-Hydroxydeoxyguanosine in Alzheimer′s disease brain tissue.
This Anti-8-Hydroxyguanosine Goat polyclonal antibody detects 8-Hydroxydeoxyguanosine in Alzheimer′s disease brain tissue. It is validated for use in ELISA and with paraffin sections.

免疫原

8-Hydroxyguanosine

應用


ELISA (≥1:250,000)
Paraffin Sections (≥1:200, proteinase K pre-treatment recommended; see figure legend, see application references)

警告

Toxicity: Standard Handling (A)

外觀

Undiluted serum.

重構

Following initial thaw, aliquot and freeze (-20°C).

分析報告

Positive Control
Alzheimer’s disease brain tissue

其他說明

Abe, T., et al. 2003. Neurosci. Lett.336, 105.
Parker, A.R., et al. 2002. Cancer Res.62, 7230.
Does not cross-react with other naturally occurring nucleotides. Antibody should be titrated for optimal results in individual systems.

法律資訊

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

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儲存類別代碼

10 - Combustible liquids

水污染物質分類（WGK）

WGK 1

閃點（°F）

Not applicable

閃點（°C）

Not applicable

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N Gueven et al.
Cell death and differentiation, 14(6), 1149-1161 (2007-03-10)
Several different autosomal recessive genetic disorders characterized by ataxia with oculomotor apraxia (AOA) have been identified with the unifying feature of defective DNA damage recognition and/or repair. We describe here the characterization of a novel form of AOA showing increased
Joseph J Boyle et al.
The American journal of pathology, 174(3), 1097-1108 (2009-02-24)
Intraplaque hemorrhage accelerates atherosclerosis via oxidant stress and contributes to lesion development and destabilization. Normally, macrophages scavenge hemoglobin-haptoglobin (HbHp) complexes via CD163, and this process provokes the secretion of the anti-inflammatory atheroprotective cytokine interleukin (IL)-10. We therefore tested the hypothesis

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