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Merck

W217018

Sigma-Aldrich

2-丁酮

FCC, FG

别名:

MEK, 乙基甲基酮, 甲基乙基酮

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About This Item

线性分子式:
C2H5COCH3
CAS号:
分子量:
72.11
FEMA號碼:
2170
Beilstein:
741880
EC號碼:
歐洲委員會號碼:
753
MDL號碼:
分類程式碼代碼:
12164502
eCl@ss:
39021202
PubChem物質ID:
Flavis號碼:
7.053
NACRES:
NA.21

生物源

synthetic

品質等級

等級

FG
Halal
Kosher

agency

meets purity specifications of JECFA

法律遵循

EU Regulation 1334/2008 & 178/2002
FCC
FDA 21 CFR 117
FDA 21 CFR 172.515
FDA 21 CFR 175.320

蒸汽密度

2.49 (vs air)

蒸汽壓力

71 mmHg ( 20 °C)

化驗

≥99.5% (GC)

形狀

liquid

自燃溫度

960 °F

expl. lim.

10.1 %

折射率

n20/D 1.379 (lit.)

bp

80 °C (lit.)

mp

−87 °C (lit.)

密度

0.805 g/mL at 25 °C (lit.)

應用

flavors and fragrances

文件

see Safety & Documentation for available documents

食物過敏原

no known allergens

感官的

ethereal

SMILES 字串

CC(CC)=O

InChI

1S/C4H8O/c1-3-4(2)5/h3H2,1-2H3

InChI 密鑰

ZWEHNKRNPOVVGH-UHFFFAOYSA-N

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象形圖

FlameExclamation mark

訊號詞

Danger

危險聲明

危險分類

Eye Irrit. 2 - Flam. Liq. 2 - STOT SE 3

標靶器官

Central nervous system

安全危害

儲存類別代碼

3 - Flammable liquids

水污染物質分類(WGK)

WGK 1

閃點(°F)

30.2 °F - closed cup

閃點(°C)

-1 °C - closed cup

個人防護裝備

Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter


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L B Goldsmith et al.
Contact dermatitis, 19(5), 348-350 (1988-11-01)
Irritant contact dermatitis along with an increased transepidermal water loss can result from exposing the skin to solvents. A study of the interaction of various solvents with human stratum corneum was made using thin-layer chromatography. Comparison of 10 solvents (trichloroethylene
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Oncogene activation in tumor cells induces broad and complex cellular changes that contribute significantly to disease initiation and progression. In melanoma, oncogenic BRAF(V600E) has been shown to drive the transcription of a specific gene signature that can promote multiple mechanisms
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T helper (TH) cell polarization during priming is modulated by a number of signals, but whether polarization to a given phenotype also influences recall responses of memory TH cells is relatively unknown. Here we show that miR-181a is selectively induced
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Kinase inhibitors block proliferative signals in BCR-ABL1+ leukemic cells, but their capacity to induce apoptosis is poorly understood. Initial studies suggested that very brief exposure to kinase inhibitors was sufficient to induce apoptosis in chronic myeloid leukemia (CML) cells. However

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