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Merck

CDS007521

Sigma-Aldrich

3-氨基-丙腈

AldrichCPR

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About This Item

经验公式(希尔记法):
C3H6N2
分子量:
70.09
EC號碼:
MDL號碼:
分類程式碼代碼:
12352200
PubChem物質ID:

形狀

liquid

SMILES 字串

NCCC#N

InChI

1S/C3H6N2/c4-2-1-3-5/h1-2,4H2

InChI 密鑰

AGSPXMVUFBBBMO-UHFFFAOYSA-N

其他說明

请注意,Sigma-Aldrich将此产品作为一系列独特化学品的一部分提供给早期发现研究人员。 Sigma-Aldrich不会收集该产品的分析数据。 买方需承担确认产品标识和/或纯度的责任。所有销售均为最终销售。

尽管SIGMA-ALDRICH的标准条款和条件以及SIGMA-ALDRICH′S和买方之间的协议中包含所有合同条款,但无论是在法律、交易过程、执行过程中的行为、以及贸易或其他方面的使用等过程中,SIGMA-ALDRICH按“原样”销售此产品并且并对此产品不作任何陈述或担保,包括:(A)适销性担保;(B)特定用途适用性担保;或(C)未侵犯第三方知识产权担保。

訊號詞

Danger

危險聲明

危險分類

Acute Tox. 4 Oral - Eye Dam. 1 - Repr. 2 - Skin Corr. 1C

儲存類別代碼

8A - Combustible corrosive hazardous materials

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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分析证书(COA)

Lot/Batch Number

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Yun-Fei Jiang et al.
Interactive cardiovascular and thoracic surgery, 28(3), 344-352 (2018-09-01)
Our goal was to examine whether interleukin-1 beta (IL-1β) originates locally and its possible relationship with matrix metalloproteinases (MMPs), apoptosis, elastin fibres and biomechanics in aortic dissecting aneurysms (DAs). Aortic DAs were induced in 24 rats with β-aminopropionitrile (BAPN); another
T Liu et al.
European review for medical and pharmacological sciences, 21(20), 4632-4639 (2017-11-14)
Elevated apoptosis of vascular smooth muscle cell (VSMC) is correlated with the occurrence of aortic dissection (AD). Yes-associated protein 1 (YAP1) is the major effector in Hippo-YAP signal pathway, which facilitates cell proliferation and suppressing apoptosis. Several studies have been
Brian L Hoh et al.
Journal of the American Heart Association, 7(8) (2018-04-15)
Estrogen deficiency is associated with the development of cerebral aneurysms; however, the mechanism remains unknown. We explored the pathway of cerebral aneurysm development by investigating the potential link between estrogen deficiency and inflammatory factors. First, we established the role of
Hiroharu Kataoka et al.
Neurologia medico-chirurgica, 60(7), 319-328 (2020-06-17)
Hemodynamic stress and chronic inflammation are closely associated with the pathogenesis of intracranial aneurysms (IAs). However, the hemodynamic and biological mechanisms triggering IA formation remain to be elucidated. To clarify them, computational fluid dynamics (CFD) and histopathological analyses in the
Taichi Ikedo et al.
World neurosurgery, 129, e361-e366 (2019-06-09)
To investigate the association between vascular morphology and the development of intracranial aneurysms (IAs), the morphological changes of intracranial arteries after IA induction were examined using a rodent model. The vascular morphology of the circle of Willis in rats was

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